Objective to investigate regulatory t cell response induced by t cell vaccination.
目的研究T细胞免疫后正常小鼠的调节性免疫应答。
Maintenance of a functional HIV-specific CD4 T cell response correlates with long term non-progression of HIV disease.
保持有效的HIV特异性CD4细胞应答与长时间非进行性HIV有关。
Combining these distinct gene-delivery vaccination approaches has the potential to induce higher levels of t cell responses and a different quality of response than either approach by itself.
联合使用这些基因表达疫苗的方法具有引导更高水平T细胞应答以及于单独使用不同的应答反应的潜力。
The idea of provoking a cell-mediated response to flu is controversial, though, in part because it takes longer to start mass-producing T-cells than antibodies.
尽管如此，这种激起细胞介导的对流感的免疫应答的想法仍存在争议，部分是因为大规模生产t细胞比抗体更耗时。
Many researchers think a T-cell response would limit disease severity but would not stop you falling ill.
许多研究者认为t细胞应答能限制疾病的严重程度，但不能预防疾病。
Treatment with IL-2, which promotes T-cell activation and proliferation, is associated with response rates of 13% to 16%.
使用白介素2治疗可以激活T细胞并使之增殖，从而使治疗有效率达13% - 16%。
Before a helper t cell can trigger a response to a pathogen or parasite, it has to "feel" these molecular bits of an invader displayed on the surface of an antigen-presenting cell (APC).
在辅助T细胞激发针对病原的免疫反应之前，它必须感觉到位于抗原递呈细胞面表的入侵者分子片段。
The magnitude and duration of T-cell immune response are critically regulated by the interaction of interleukin-2 (IL-2) and its high-affinity receptor (IL-2R) on the surface of the cell.
白细胞介素2 (IL - 2)与其高亲和力受体(IL - 2r)的相互作用在调节T细胞免疫反应的强度和持续时间方面起着关键作用。
Conclusion TJU103 is capable of markedly inhibiting t cell proliferative response in vitro and can decrease GVHD incidence after allogeneic stem cell transplantation in mice.
结论TJU103不仅可以显著抑制体外t淋巴细胞增殖效应，而且可显著降低小鼠同种异基因造血干细胞移植模型GVHD的发生率。
Blood tests taken throughout the experiment showed that IL-7 seemed to switch off the production of SOCS3, thereby ramping up the T-cell response.
贯穿本实验始终的血液测试显示：IL - 7看来关闭了SOCS3的生产过程，从而增强了T细胞的响应水平。
They are able to suppress allogenic T-cell response and modify maturation of antigen-presenting cells.
其能够抑制同种异体的T细胞的应答和修饰抗原呈递细胞的成熟。
These findings indicate that, in contrast to derivatives of ESCs, abnormal gene expression in some cells differentiated from iPSCs can induce T-cell-dependent immune response in syngeneic recipients.
这些发现说明，相对于胚胎干细胞的后代，从诱导多能干细胞分化来的某些细胞中异常的基因表达会在同基因的受体身上引起T细胞依赖的免疫反应。
It is very crucial to identify antigen peptide recognized by t cell to study adaptive immune response and immune regulation.
确定T细胞所识别抗原分子上的短肽序列对T细胞表位进行定位，对于研究特异性免疫应答有着重要意义。
Liver injury is associated with T-cell-mediated immune response indicating immune-mediated liver injury.
肝损伤同T细胞免疫反应相关，标志免疫反应造成肝损伤。
CD4+CD25+ regulatory T cell is a crowd of cells, which can regulate and suppress the immune response.
CD4+CD2+5调节性T细胞是一群具有免疫调节或免疫抑制功能的细胞。
SOCS1 silent DC could promote DC maturity and enhance the antitumor response of t cell.
SOCS1沉默的树突状细胞能够促进自身成熟并增强其诱导的T细胞的抗肿瘤活性。
SOCS1 silent DC could promote DC maturity and enhance the antitumor response of t cell.
SOCS1沉默的树突状细胞能够促进自身成熟并增强其诱导的T细胞的抗肿瘤活性。
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