Conclusions t pa and pai 1 not only participate in the pathophysiological changes of peripheral nerves after crush injury, but have some effects on peripheral nerve repair as well.
结论tPA和PAI1参与周围神经挤压伤后的病理生理改变，在损伤神经修复过程中具有一定的作用。
Objective To assess the clinical significance of plasma t-PA, PAI-1 in acute cerebral infarction.
目的探讨血浆组织型纤溶酶原激活物(t -PA)及其抑制物(PAI - 1)在急性脑梗死发病中的意义。
Results: The changes of high TG, PAI-1 and low level t-pA were most significant abnormal charctor of Cerbral Infarction.
结果：血高TG、高PAI 1，与低tPA含量变化为脑梗塞患者异常变化特征，与正常组相比有显著性差异（P。
Objective the effect of vascular smooth muscle cells (VSMCs) on the abilities of secreting t-PA and PAI-1 by th cultured endothelial cells (ECs) under shear stress has been studied.
目的探讨切应力作用下联合培养的血管平滑肌细胞对内皮细胞分泌t - PA和PAI - 1的影响。
Results: The activity of PAI-1 was higher and t-PA was lower in patients with coronary heart disease, and this difference is also found between Type a and Non Type a patients.
结果：冠心病患者比正常人群的PAI - 1活性上升，t -PA活性下降；这种改变在不同行为类型之间比较，A型行为类型比非a型行为类型的冠心病患者PAI - 1的活性升高而t - PA活性降低。
Objective To investigate effects of fibrinolytic system upon deep venous thrombosis, focused on PLG, t-PA and PAI-1.
目的以纤溶酶原、t - PA和PAI - 1为轴心，探讨纤溶系统对深静脉血栓形成(DVT)的影响。
Conclusion the unbalance between t-PA and PAI can promote the formation of micro-thrombus and further exacerbate high pulmonary pressure.
结论t - PA及PAI关系失衡，有助于动脉内微血栓形成，促进或加重肺动脉高压的形成。
Conclusion the unbalance between t-PA and PAI can promote the formation of micro-thrombus and further exacerbate high pulmonary pressure.
结论t - PA及PAI关系失衡，有助于动脉内微血栓形成，促进或加重肺动脉高压的形成。
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