Aim: To observe the differences of hemodynamics and nitric oxide synthase (NOS) activity of ventricular cardiac muscle in two septic shock models and explore the possible mechanism.
目的观察两种败血症休克模型大鼠的血流动力学及心肌细胞一氧化氮合酶活性变化的异同，探讨一氧化氮合酶参与败血症休克性心肌抑制的机制。
Aim: To observe the differences of hemodynamics and nitric oxide synthase (NOS) activity of ventricular cardiac muscle in two septic shock models and explore the possible mechanism.
目的观察两种败血症休克模型大鼠的血流动力学及心肌细胞一氧化氮合酶活性变化的异同，探讨一氧化氮合酶参与败血症休克性心肌抑制的机制。
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