To understand further the molecular mechanism of its function, human neuroblastoma SK N SH cell line was chosen as a model to study its signal transduction pathway.
为了进一步阐明其作用的分子机制，以SKNSH成神经瘤细胞（SK细胞）为模型进行了研究。
Methods: By using patch clamp technique, measure voltage gated calcium currents in undifferentiated SH SY5Y cell line.
方法：通过膜片钳手段，测量未分化的SHSY5Y细胞钙离子通道电流；
The mechanisms are probably that zinc can regulate the receptor of liver cell for HDL-C and LDL-C, compete with arsenic for -SH binding and precipitate the excretion of arsenic.
可能与锌调节肝细胞相应受体、与砷竞争性结合琉基、促进砷的排泄有关。
Methods the SH-SY5Y cells were treated with different concentrations of dopamine, and cell viability, as well as the marker of oxidative stress, was observed in a time course.
方法用不同浓度的多巴胺处理SH -SY5Y细胞，在一定时程内的不同时间点观察细胞活性，各种氧化应激指标。
Conclusion The anticomplementary polyphenol XCG-7 from the seed of Toona sinensis can promote SH-SY5Y cell growth, and protect SH-SY5Y cells from complement-induced injury.
结论香椿果抗补体活性多酚XCG-7能促进SH-SY5Y细胞的生长，对补体损伤神经细胞具有一定的保护作用。
SH-SY5Y cells are an adrenergic clone of the human neuroblastoma cell line SK-N-SH and a very useful model for the study of neuronal cell terminal differentiation.
SY5Y细胞是人神经母细胞瘤细胞系SK-N-SH中的一个克隆，它是研究神经细胞终末分化的一个极有用的模型。
The influence of KCl on the release of glutamic acid from culture human neuroblastoma cell line (SH SY5Y) was observed by using HPLC.
利用高效液相方法观察了氯化钾对培养人神经母细胞瘤细胞系(SHSY5Y)细胞谷氨酸释放的影响。
Maltol was shown to protect sh SY5Y cell from oxidative damage and maintain its normal functions.
结果证明，麦芽酚可以有效保护活性氧对神经细胞的氧化损伤，维持细胞的正常生理功能。
Maltol was shown to protect sh SY5Y cell from oxidative damage and maintain its normal functions.
结果证明，麦芽酚可以有效保护活性氧对神经细胞的氧化损伤，维持细胞的正常生理功能。
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