• Is cortisol necessary for severe sepsis or septic shock?

    严重脓毒症及感染性休克时是否需用皮质醇?

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  • Different doses of HC in early septic shock were observed.

    观察不同剂量HC对早期脓毒性休克的作用。

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  • All of the septic shock patients were free from heart or renal failure.

    所有的感染性休克患者没有心脏或者肾脏衰竭。

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  • If left untreated, the bacterial blood condition can develop into septic shock.

    如果不及时治疗,血中细菌可能会导致感染性休克。

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  • Persistent bacteremia often leads to severe sepsis or overwhelming septic shock.

    持续性的菌血症经常导致严重的败血症,甚至恶化为败血性休克。

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  • Objective: Investigated the effect of lactate-directed treatment on septic shock.

    目的:探讨血乳酸目标指导性治疗对于感染性休克的治疗效果。

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  • In this case, because it is caused by bacterial toxins, it is called septic shock.

    在这种情况下,因为它是造成细菌毒素,这是所谓的感染性休克。

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  • Without immediate treatment, septic shock follows, with a mortality rate over 50%.

    如不及时处理,败血症会恶化成败血性休克,其死亡率高达50%以上。

    youdao

  • Septic shock and multi organ failure were the predicting factors of poor prognosis.

    感染性休克与多脏器功能衰竭是预后不良的先兆因素。

    youdao

  • Multi-organ failure and septic shock were the predicting factors of poor prognosis.

    多脏器功能衰竭与感染性休克是预后不良的先兆因素。

    youdao

  • Blood gas analysis is of great value to early diagnosis and treatment of septic shock.

    血气分析对感染性休克的早期诊断和临床治疗具有重要的临床价值。

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  • Septic shock and multisystem organ failure (MSOF) are main lethal factors in aged AOSC.

    感染性休克和多系统器官衰竭(MSOF)是老年aosc死亡的主要原因。

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  • Objective To investigate the clinic value of interleukin-6 (IL-6) in sepsis and septic shock.

    素-6(IL-6)在新生儿败血症及败血症休克中的变化及临床意义。

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  • Septic shock, one of the most severe complication, is deadly, its mortality varies from 35%-70%.

    脓毒症休克这一最严重的并发症是致命性的,死亡率达35-70%。

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  • Results 89 cases of patients cured, 11 improved, one cases of deaths, died of severe septic shock.

    结果本组病例治愈89例,好转11例,死亡1例,死于重度感染性休克。

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  • Clinically, septic shock caused by sepsis is a frequent cause of death for patients in intensive care.

    在临床上,因脓毒血症所导致的脓毒症性休克是重症监护患者最常见的死亡原因。

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  • However, not everyone is convinced of the predictive value of BNP levels in patients with septic shock.

    然而,并不是每个人都相信能预测感染性休克患者BNP水平。

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  • Methods Make a retrospective analyse to one case with serious septic shock after liver transplantation.

    方法回顾性分析一例肝移植术后因严重感染性休克行持续性血液滤过的患者。

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  • That is probably also why protein C supplementation is such a promising potential therapy for septic shock.

    这也是补充蛋白C是治疗内毒素休克一项极具希望疗法的所在。

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  • CONCLUSION Penehvclidine Hydrochloride would be one of the ideal vasoactive drugs in treatment for septic shock.

    结论长托宁是脓毒性休克患者较理想的血管活性药。

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  • Would decrease mortality among patients with septic shock who were being treated with conventional vasopressor …

    低剂量的传统血管加压素治疗可以降低败血症病人的死亡率…

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  • ObjectiveTo evaluate clinical significance of the monitoring of oxygen metabolic variables in septic shock patients.

    目的评价脓毒性休克患者氧代谢参数监测的临床意义。

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  • Septic shock (36.2%) and heart failure (20.6%) were major lethal causes, while only 14.6% died of respiratory failure.

    脓毒性休克(36.2%)与心功能衰竭(20.6%)是主要死亡原因,仅14.6%患者死于呼吸衰竭。

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  • Conclusion: lactate-directed treatment greatly improves the physiological status and mortality of septic shock patients.

    结论:血乳酸目标指导性治疗可改善感染性休克患者的生理指标和死亡率。

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  • This gene codes for a receptor - nicknamed a Toll-like receptor - that binds to a bacterial product involved in septic shock.

    这个基因编码一个受体,称为Toll样受体,这种受体可以结合到参与感染性休克的细菌产物上。

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  • PDS can improve hypotension in septic shock rats, inhibit NOS activity and reduce NO content by inhibiting its production.

    结论PDS能够明显改善内毒素休克大鼠的低血压状态,降低NOS活性、NO含量,并对其NO的生成具有抑制作用。

    youdao

  • PDS can improve hypotension in septic shock rats, inhibit NOS activity and reduce NO content by inhibiting its production.

    结论PDS能够明显改善内毒素休克大鼠的低血压状态,降低NOS活性、NO含量,并对其NO的生成具有抑制作用。

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