Aortic thrombi and renal ischemia have also been described.
亦有主动脉血栓及肾脏缺血之报告。
Protective effect of panax notoginsenosides on renal ischemia reperfusion injury in rats;
目的观察三七总苷对大鼠肾缺血再灌注损伤的保护作用。
NO may involve in the influence of the renal ischemia-reperfusion injury on the cerebral function.
NO可能参与了肾缺血-再灌注损伤对脑功能的影响。
Objective To investigate the protective effects of ginsenosides (GS) on renal ischemia reperfusion injury in rats.
目的观察研究人参总皂甙(GS)对肾缺血再灌注损伤的保护作用。
To investigate the expression of intercellular adhesive molecular 1 (ICAM 1) in renal ischemia reperfusion injury.
观察肾缺血再灌注过程中，细胞间粘附分子1 (ICAM 1)在肾组织中的表达。
With renal ischemia and reperfusion, the SOD activities in renal tissue declined and the MDA content significantly increased.
给人参总皂甙后能显著减少肾组织中MDA含量，提高SOD活性及内生肌酐清除值。
Objective To investigate the effects of renal ischemia reperfusion on renal function and renal vascular responsibility in rats.
目的探讨肾缺血再灌注对大鼠肾功能和肾血管反应性的影响。
Intraoperative renal ischemia time, postoperative complications and incidence of postoperative local recurrence were evaluated.
评估肾蒂阻断时间、术后并发症及局部复发情况。
Conclusions: tea polyphenols preconditioning group to kidney could protect the kidney against renal ischemia reperfusion injury.
结论：在肾脏缺血再灌注损伤过程中，茶多酚能起到对肾脏的保护作用。
Objective To investigate the effect of Astragalus on expression of intercellular adhesive molecule 1 (ICAM 1) in renal ischemia reperfusion injury.
目的探讨黄芪在肾缺血再灌注损伤中对细胞粘附分子1（ICAM 1）表达的影响。
The results are as follows:1. RBF is decreased by electroacupuncturing "Shenshu" point under both conditions of normal and glycerol-induced renal ischemia.
结果显示：在双侧肾神经完整的情况下，无论是正常，还是肾缺血状态，电针“肾俞”穴均可使RBF下降；
The mechanism of the acute tubular necrosis (ATN) caused by acute renal ischemia-reperfusion injury is complicated and there are many correlation factors in it.
急性缺血再灌注引起急性肾小管坏死发生机制较为复杂，相关因素比较多。
Objective to explore the effects of hypertonic saline preconditioning on the expression of intercellular adhesion molecules-1 (ICAM1) after renal ischemia-reperfusion injury in rats.
目的探讨高渗盐液预处理对大鼠肾缺血再灌注损伤后肾组织细胞间黏附分子- 1的表达的影响。
Animal ARF can be induced by renal ischemia-reperfusion injury, glycerine, gentamicin and mercuric chloride, and typical acute tubular necrosis can be observed in pathological changes.
肾动脉钳闭引起的肾脏缺血再灌注以及甘油、庆大霉素、氯化汞均可致实验动物急性肾衰竭，病理学上可见典型的急性肾小管坏死。
These results suggest that oxygen free radicals may damage the activity of cytochrome oxidase, the reduction of which may play an important role in the injury of renal ischemia and reperfusion.
提示氧自由基可能损害细胞色素氧化酶活性，细胞色素氧化酶活性降低在肾缺血再灌流损伤中可能起重要作用。
Purpose: To investigate the role of heat shock protein 70 (HSP70) in renal ischemia-reperfusion injury in rats after ischemic preconditioning by examining the change of the expression of HSP70.
目的：研究缺血预处理对肾脏缺血再灌注损伤的保护作用，观察热休克蛋白70 (HSP70)的表达变化并探讨其作用机制。
Objective: to observe the influence of Astragalus injection on the apoptosis of cells in rats after renal ischemia reperfusion injury in order to elucidate the mechanism of its protective effect.
目的：观察黄芪注射液对大鼠肾脏缺血再灌注细胞凋亡的影响，探讨其对肾脏缺血再灌注损伤的保护机制。
Objective To evaluate the functional alternations of canine renal ischemia and reperfusion injury by using MR perfusion-weighted imaging, and to correlate the imaging with the pathologic findings.
目的结合病理对照研究MR灌注成像评价犬肾缺血再灌注损伤后肾功能的变化。
Renal injury after ischemia appears to be a consequence of tissue hypoxia not only from interrupted blood supply but also from the process of reperfusion which leads to an active inflammatory process.
肾缺血后损伤显示，损伤不仅是血供中断后组织缺氧的结果，更有再灌注进程中引起炎症反应的激活过程。
The histopathological changes of the kidneys included renal tubular necrosis and detachment, formation of casts, glomerular ischemia and shrinkage and infiltration of large amount of PMN in LPS group.
而肾组织的病理改变主要表现为肾小管的坏死、脱落，管型形成，肾小球缺血、皱缩，肾组织内见多量pmn滞留。
Conclusion PROG may exacerbate the renal injury of ischemia-reperfusion.
结论黄体酮可能加剧肾缺血-再灌注损伤。
OBJECTIVE To investigate the protective mechanism of adenosine in renal damage of ischemia reperfusion.
探讨腺苷对肾脏缺血再灌注损伤的保护作用机制。
Because of high consumption of oxygen and large blood flow in the renal tissues, kidney is the most sensitive organ to ischemia. So the renal injury happens as a high incidence.
由于肾组织能量消耗大，肾血流量多，使其成为对缺血缺氧最敏感的器官。
This is an acute renal infarction. Note the wedge shape of this zone of coagulative necrosis resulting from loss of blood supply with resultant tissue ischemia that produces the pale infarct.
急性肾梗死，属凝固性坏死，坏死区域呈楔形，是由于组织血供减少引起缺血，终而产生灰白的梗死区。
Conclusion There are failure of mesenteric microcirculation during renal poster ischemia reperfusion injure.
结论兔肾脏缺血再灌注时存在肠微循环障碍。
The measures of protecting the renal function were adopted, if operated kidney had been in heat ischemia for a long time in the operation.
当术中手术肾热缺血时间过长时，需采用保护肾功能措施。
Objective To discuss the ultrastructure changes of renal microcirculation while acute cerebral ischemia occurs.
目的探讨急性脑缺血时肾微循环的超微结构改变。
Objective To investigate the effects of renal ischemic postconditioning(Renal PostC) on myocardium enzymes in rabbits with acute myocardial ischemia and reperfusion.
目的：观察白附片对正常大鼠和肾阳虚证大鼠心肌酶的影响，探索其对两种大鼠心脏毒性的差异。
Objective To investigate the effects of renal ischemic postconditioning(Renal PostC) on myocardium enzymes in rabbits with acute myocardial ischemia and reperfusion.
目的：观察白附片对正常大鼠和肾阳虚证大鼠心肌酶的影响，探索其对两种大鼠心脏毒性的差异。
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