Conclusion in the process of focal cerebral ischemia and reperfusion, regulating the quantity of NO in the propriety range may protect and avoid purpose of neuronal cell injury.
结论局灶性脑缺血后，调节NO生成量在适当范围，可以达到保护、防止神经细胞进一步损伤的目的。
Conclusion in the process of focal cerebral ischemia and reperfusion, regulating the quantity of NO in the propriety range may protect and avoid purpose of neuronal cell injury.
结论局灶性脑缺血后，调节NO生成量在适当范围，可以达到保护、防止神经细胞进一步损伤的目的。
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