Objective:To study the mechanisms underlying N methyl D aspartate (NMDA) induced excitoprotection in cultured rat cerebellar granule neurons.
目的：研究N甲基D天冬氨酸（NMDA）诱导大鼠小脑颗粒神经元兴奋毒性保护作用的机制。
Objective to investigate the neurotoxic effect of N-methyl-D-aspartate on neurons of retinal ganglion cell layer in rat.
目的探讨n -甲基- D -天冬氨酸(NMDA)对大鼠视网膜神经节细胞层(RG CL)神经元的毒性作用。
Objective: To study the dynamic changes of nitric oxide (n o) and nitric oxide synthase (NOS) in rat brain tissues after experimental brain trauma and their significance.
目的：研究实验性颅脑伤大鼠脑组织一氧化氮(NO)和一氧化氮合酶(NOS)含量的动态变化及其意义。
Objective To study the changes of N methyl D aspartate(NMDA) receptor in the rat brain during reperfusion after local ischemia.
目的研究大鼠脑局灶性缺血再灌注模型中N甲基D天冬氨酸（NMDA）受体随时间的变化。
Collecting all the results, impair N-cadherin-medianted adhesion increasing the susceptibility of isolated rat hearts probably through affecting the gap junctions in the intercalated discs.
以上实验结果，显示削弱n -钙粘蛋白的粘附功能可能通过影响缝隙连接的功能增加心律失常发生的易感性。
Methods: Primary cultured neonatal rat myocardial cell were randomly divided into five group: normal control group (n), hypoxic group (h), AM 1 (A1), AM 2 (A2), AM 3 (A3).
方法：将原代培养的乳鼠心肌细胞随机分为五组：正常对照组(N组)、缺氧对照组(H组)、缺氧加黄芪组1 (A 1组)、缺氧加黄芪组2 (A 2组)、缺氧加黄芪组3 (A 3组)。
Methods: Primary cultured neonatal rat myocardial cell were randomly divided into five group: normal control group (n), hypoxic group (h), AM 1 (A1), AM 2 (A2), AM 3 (A3).
方法：将原代培养的乳鼠心肌细胞随机分为五组：正常对照组(N组)、缺氧对照组(H组)、缺氧加黄芪组1 (A 1组)、缺氧加黄芪组2 (A 2组)、缺氧加黄芪组3 (A 3组)。
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