Laser confocal microscopy was used to observe the locations of Mfn2 gene with and without PKA in cells.
激光共聚焦显微镜观察完整的和去除蛋白激酶A磷酸化位点的线粒体融合素2基因在细胞中的定位。
CONCLUSION: PKC, instead of PKA, was involved in the signal transduction of the rapid effect of B on glycine uptake in C6 cells.
结论：上述结果提示蛋白激酶C参与了硫酸皮质酮快速抑制C6细胞摄取甘氨酸的信号转导过程。
In the paper, a microtiter plate method was used to determine the residual prekallikrein activator(PKA) activity in blood products.
本文应用微量滴定板方法测定血液制品中残留的PKA活性。
So this PKA sensor not only indicates whether the enzyme is on or off, but also locates where PKA is being turned on or off within the cell.
所以这种PK A传感器不仅显示酶是否开或关，而且当PKA正开或关时在胞内定位它。
CONCLUSION: PKA CREB signal transduction is probably involved in the spatial learning and memory function of the MrD and hippocampus in rats.
结论：PK A- CREB信号通路可能参与了大鼠纹状体边缘区及海马的空间学习记忆功能。
When PKA is turned on and is physically close to a biosensor, PKA itself changes the shape of the biosensor, causing it to glow green instead.
当PKA开启时并且有酶分子贴近生物传感器时，PKA会改变生物传感器的形状，导致生物传感器发稳定的绿光。
Objective To investigate the effects of increased protein kinase a (PKA) activity on the proliferation of T24 cell line and the possible mechanism.
目的探讨蛋白激酶A (pka)活性增高对膀胱癌细胞株t24增殖的影响及其机制。
The effective mobility (meff) of ten model dipeptides was measured by CZE at different pH values, enabling to determine their pKa values, charge and meff at any pH.
通过CZE在不同的PH值下测得了十个模式二肽的有效迁移率，从而能够确定在每一PH值下它们的解离常数值，电荷情况及有效迁移率。
Objective:To observe the changes of the activity of protein kinase A(PKA) and C(PKC), and further investigate the mechanisms of anti-hypothermia action of Guizhi Tang.
目的：研究低体温大鼠下丘脑蛋白激酶A（PKA）、蛋白激酶C（PKC）活性的变化及桂枝汤的升温作用机理。
The assay begins with a standard kinase reaction performed in the provided reaction Buffer with a provided Bisamide Rhodamine 110 peptide substrate (PKA R110 substrate).
检测开始时、先用试剂盒提供的PKA双酰胺罗丹明110肽底物进行一个标准的激酶反应。
AIM: To examine possible direct effects of the vasodilators hydralazine and KRN2391 on the activities of protein kinase A (PKA) , protein kinase G (PKG), and protein kinase C (PKC).
目的：研究血管扩张药肼酞嗪和KRN2391对蛋白激酶的直接作用以探讨其作用机制。
Evidence suggests that PKA regulates lipolysis by phosphorylating perilipin (13-17), resulting in a conformational change that exposes lipid droplets to endogenous, hormone-sensitive lipases (14).
已有证据显示PKA通过磷酸化脂周素调节脂解（13-17），引起构象改变使脂滴暴露给内源性的，激素敏感的脂肪酶（14）。
Evidence suggests that PKA regulates lipolysis by phosphorylating perilipin (13-17), resulting in a conformational change that exposes lipid droplets to endogenous, hormone-sensitive lipases (14).
已有证据显示PKA通过磷酸化脂周素调节脂解（13-17），引起构象改变使脂滴暴露给内源性的，激素敏感的脂肪酶（14）。
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