Objective To investigate the roles of PI3K pathway in multicellular resistance of lung adenocarcinoma A549 cells.
目的探讨PI3K信号通路在肿瘤群集耐药中的作用。
Conclusion: Both spinal and supraspinal neuronal Phosphoinositide 3-kinase (PI3K) pathway played important role in morphine dependent and naloxone-precipitated withdrawal response in mice.
结论：脊髓和脊髓上中枢神经元pi - 3 K信号通路均参与吗啡依赖的形成及戒断反应的表达。
Conclusion the PI3K signal pathway may be involved in AHR and the airway remodeling through promoting airway smooth muscle cells proliferation in asthma.
结论PI 3 K信号通路可能通过促进气道平滑肌细胞增殖参与哮喘气道高反应性及气道重构过程。
Conclusions FAK and PI3K signal pathway induced by OPN should participate in invasion and metastasis of later carcinoma of large intestine.
结论O PN诱导的FAK和PI 3 K信号途径参与了大肠癌中晚期发生的侵袭与转移过程。
This data complements Drosophila genetics studies suggesting the possible involvement of the tuberin-hamartin complex in the PI3K/Akt mediated insulin pathway (8-10).
这些数据和果蝇（Drosophila）遗传学研究表明tuberin-hamartin 复合物可能在PI3K/AKT介导的胰岛素通路中取作用（8-10）。
Previous data showed that NOK could activate the phosphatidylinositol 3 -kinase (PI3K) pathway in stable BaF3 cells.
前期的研究表明，NOK在小鼠前B细胞(BaF3)中能够激活磷脂酰肌醇3-激酶(PI3K)信号通路。
Previous data showed that NOK could activate the phosphatidylinositol 3 -kinase (PI3K) pathway in stable BaF3 cells.
前期的研究表明，NOK在小鼠前B细胞(BaF3)中能够激活磷脂酰肌醇3-激酶(PI3K)信号通路。
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