Gallium can alleviate the damage of osteocyte ultrastructure elicited by osteoporosis.
结论镓盐对骨质疏松引起的骨细胞超微结构的损伤有改善作用。
Objective to investigate the influence of gallium on osteocyte ultrastructure in osteoporosis.
目的探讨镓盐对骨质疏松骨细胞超微结构的影响。
Conclusion Gallium can alleviate the damage of osteocyte ultrastructure elicited by osteoporosis.
目的探讨镓盐对骨质疏松骨细胞超微结构的影响。
Conclusion Gallium can alleviate the damage of osteocyte ultrastructure elicited by osteoporosis.
结论镓盐对骨质疏松引起的骨细胞超微结构的损伤有改善作用。
At postoperative 7 week, the humerus sample showed that osteocyte separated from osseous lacuna, some nucleus pycnosis and rough endoplasmic hollowed;
术后7周实验组肱骨标本可见骨细胞与骨陷窝分离，有的细胞核固缩粗面内质网肿胀空化；
AIM to investigate osteocyte apoptosis in the trabecular bone of rats with spinal cord injury (SCI) and its role in the pathogenesis of osteoporosis following SCI.
目的探讨脊髓损伤(SCI)对骨细胞凋亡的影响及其在SCI后骨质疏松发病中的可能机制。
To understand how Lrp5 influences bone properties, we generated mice with osteocyte-specific expression of inducible Lrp5 mutations that cause high and low bone mass phenotypes in humans.
为了研究Lrp5如何影响骨特性，我们建立了骨细胞特异性表达诱导型Lrp5基因突变的小鼠，该基因突变可导致人类高和低骨量表型。
To understand how Lrp5 influences bone properties, we generated mice with osteocyte-specific expression of inducible Lrp5 mutations that cause high and low bone mass phenotypes in humans.
为了研究Lrp5如何影响骨特性，我们建立了骨细胞特异性表达诱导型Lrp5基因突变的小鼠，该基因突变可导致人类高和低骨量表型。
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