Analyzing brain cells from normal mice.
分析正常小鼠的脑细胞。
They compared the so-called sweet-blind mice to normal mice.
他们用这种“甜味盲”老鼠与正常老鼠做比较。
The mutant mice also bounced back faster than the normal mice from sleep deprivation.
在被剥夺了睡眠后，变异老鼠比正常老鼠精神恢复得更快。
As a result, they are able to eat large amounts of food yet weigh less than normal mice.
结果就是，它们吃大量食物，却比正常老鼠要轻。
Their metabolism remained high despite being less hungry and not eating as much as normal mice.
虽然吃的不像正常老鼠那么多，也不那么饥饿，这些老鼠的新陈代谢依然旺盛。
The mice with the mutation needed less sleep than normal mice.
携带有突变基因的小鼠比正常鼠需要较少的睡眠。
Another 8 normal mice were chosen as group 5 (blank control group).
另8只正常小鼠为第5组(空白对照组)。
Methods: Gastrointestinal propulsion rate was taken as an observed index in normal mice.
方法：以正常小鼠为观察对象，以胃肠推进率为观察指标。
So the MCP can improve NK cell activity and has no immunosuppressed functions in normal mice.
因此，冬寒菜粉可提高正常小鼠NK细胞活性，而对其无免疫抑制作用。
They had a condition called progeria, meaning that they aged much more rapidly than normal mice.
它们的症状也称早老症，意味着它们比一般的小白鼠衰老的更快。
The effects of RHPS & APS on immune function in normal mice and immunosuppressed mice were studied.
红芪多糖（RHPS）和黄芪多糖（APS）对正常小鼠和免疫抑制小鼠的免疫功能有影响。
Immune cell Numbers in these mice skyrocketed - T-cells increased sixfold compared with normal mice.
这些小鼠中的免疫细胞数量激增，其中T细胞与正常小鼠相比增加了六倍。
Even after only 7 days of leucine deprivation, the normal mice lost 50 percent of their fatty tissue.
只在缺乏亮氨酸的饮食7天之后，正常小鼠就减少了50%的脂肪组织。
These findings suggested that unlike normal mice, the obese mice lacked a hormone that made them feel full.
这些发现表示，与正常老鼠不同，肥胖老鼠缺少让它们感觉吃饱的一种激素。
After being injected, these mice, who also were memory-impaired, performed as well as normal mice in memory tests.
注射完以后，这些原来记忆力受损的老鼠在记忆测试中，表现和正常老鼠一样。
Results in normal mice, perforin was negative and connexin 43 was located in the intercalated disks of cardiac muscle.
结果：正常小鼠心肌中未见穿孔素阳性表达，连接蛋白43在心肌闰盘中呈阳性表达。
Effect of Xiao Tang 2 and Xiao Tang 5 blood glucose in diabetes and normal mice and on glucose tolerance of mice was observed.
观察消糖2号和消糖5号对糖尿病小鼠血糖、对正常小鼠血糖及糖耐量的影响。
Studies in the past have shown that male mice created without NPY are more anxious than normal mice, which is hardly surprising.
过去的研究已经表明，没有神经肽y的雄性小鼠比正常小鼠更容易焦虑，这并不足以令人惊奇。
These mice ended up getting sick a lot, and needed to eat much more than normal mice with intestinal bacteria in order to gain weight.
结果是，这些基因变异的老鼠不仅经常生病，而且要比正常老鼠吃更多才能长肥。
But they didn't know how the animals accomplished this feat or whether the cancer resistance could be transferred to normal mice.
但他们尚未知道这些动物如何获得这种能力，这种抗癌能力是否可以转移给正常小鼠也未可知。
Methods Phosphoproteins were extracted from lysates of normal mice livers by phosphate metal affinity chromatography (PMAC) resin.
方法取正常小鼠肝脏，裂解肝脏后利用磷酸盐金属亲和层析(PMAC)树脂提取磷酸化蛋白。
Compared with the normal mice, those with one mutant gene slept about 1.2 hours less, and mice with two mutant genes slept 2.5 hours less.
与正常的老鼠相比，携带一个变异基因的老鼠睡眠时间少1.2小时，而携带有2个变异基因的老鼠睡眠时间少2.5小时。
However, after four days of oral treatment with NAP and SAL, the Down's mice learned to navigate the maze just as easily as normal mice.
然而，口服NAP和SAL治疗四天后，唐氏模型鼠可以像正常鼠一样容易地学习适应水迷宫。
Objective:To study the effects of Jiangtang Mixture on blood glucose and blood lipid in normal mice and diabetic mice induced by alloxan.
目的：观察分析降糖合剂对正常小鼠血糖及四氧嘧啶糖尿病小鼠血糖、血脂的影响。
In mice injected with human bladder, breast, lung, or melanoma cancer cells, PL inhibited tumor growth but showed no toxicity in normal mice.
注射人膀胱癌细胞、乳腺癌细胞、肺癌细胞或者黑色素瘤癌细胞的小鼠，给予PL可以抑制这些肿瘤的生长，但PL对正常小鼠未见毒性作用。
Results: Huangqi inhibited the gastric emptying in normal mice and significantly antagonized the delayed gastric emptying induced by atropine.
结果发现黄芪可抑制正常小鼠的胃排空，对阿托品所致的胃排空减缓具有一定的拮抗作用。
There was no significant difference of the expression level of serum IL-10 between H22 tumor-bearing mice and treatment mice with the normal mice.
H22荷瘤小鼠和五味子多糖给药组小鼠血清IL-10的表达水平与正常小鼠间无显著差异。
A new study confirms that anesthesia is safe for normal mice but potentially harmful for mice with genetic risk factors for Alzheimer's disease (AD).
一项新研究证实了麻醉对于正常小鼠来说是安全的，但是对于有AD遗传危险因素的小鼠是有潜在危险性的。
Objective: To research on the metabolism of ALA taking orally in lung of normal mice and seek suitable dose and diagnostic time of ALA taking orally.
目的：探求ALA口服在正常小鼠肺组织中的代谢，寻求肺组织光敏诊断最佳的ALA口服剂量及时间。
Objective: To research on the metabolism of ALA taking orally in lung of normal mice and seek suitable dose and diagnostic time of ALA taking orally.
目的：探求ALA口服在正常小鼠肺组织中的代谢，寻求肺组织光敏诊断最佳的ALA口服剂量及时间。
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