Analyzing brain cells from normal mice.
分析正常小鼠的脑细胞。
They compared the so-called sweet-blind mice to normal mice.
他们用这种“甜味盲”老鼠与正常老鼠做比较。
"Normal mice treated with the drugs dropped some weight," he said.
“正常老鼠在吃了药之后体重减轻了，”他说。
The mice with the mutation needed less sleep than normal mice.
携带有突变基因的小鼠比正常鼠需要较少的睡眠。
The mutant mice also bounced back faster than the normal mice from sleep deprivation.
在被剥夺了睡眠后，变异老鼠比正常老鼠精神恢复得更快。
As a result, they are able to eat large amounts of food yet weigh less than normal mice.
结果就是，它们吃大量食物，却比正常老鼠要轻。
When the team fed the mice high-fat diet, all grew obese, as did a group of normal mice.
当小组给小鼠喂饲高脂食物，一组正常小鼠全部变成肥胖。
Methods: Gastrointestinal propulsion rate was taken as an observed index in normal mice.
方法：以正常小鼠为观察对象，以胃肠推进率为观察指标。
Their metabolism remained high despite being less hungry and not eating as much as normal mice.
虽然吃的不像正常老鼠那么多，也不那么饥饿，这些老鼠的新陈代谢依然旺盛。
These mice had double the amount of inflammation in response to CGRP than did normal mice.
这些小鼠对CGRP的炎症反应双倍于正常小鼠。
So the MCP can improve NK cell activity and has no immunosuppressed functions in normal mice.
因此，冬寒菜粉可提高正常小鼠NK细胞活性，而对其无免疫抑制作用。
The super-stressed mice that couldn’t grow new neurons gave up swimming faster than the normal mice.
这些不可再生神经元的过度压力小老鼠们比正常的小老鼠们先停止的游泳。
The super-stressed mice that couldn't grow new neurons gave up swimming faster than the normal mice.
这些不可再生神经元的过度压力小老鼠们比正常的小老鼠们先停止的游泳。
They had a condition called progeria, meaning that they aged much more rapidly than normal mice.
它们的症状也称早老症，意味着它们比一般的小白鼠衰老的更快。
The effects of RHPS & APS on immune function in normal mice and immunosuppressed mice were studied.
红芪多糖（RHPS）和黄芪多糖（APS）对正常小鼠和免疫抑制小鼠的免疫功能有影响。
Even after only 7 days of leucine deprivation, the normal mice lost 50 percent of their fatty tissue.
只在缺乏亮氨酸的饮食7天之后，正常小鼠就减少了50%的脂肪组织。
These findings suggested that unlike normal mice, the obese mice lacked a hormone that made them feel full.
这些发现表示，与正常老鼠不同，肥胖老鼠缺少让它们感觉吃饱的一种激素。
But when they were subjected to stress, the level of stress hormones remained high much longer than in normal mice.
但它们感受到了压力的时候，它们的应激激素会持续比正常小老鼠的要偏高并且持续更久些。
They set up an experiment to compare normal mice that produce the binding proteins with mice engineered to lack them.
他们设立了一个不能合成绑定蛋白的老鼠的实验组来对照正常的能够合成绑定蛋白的老鼠们。
Nutritional blood flow of normal mice and the mice of myocardial ischemia induced by pituitrin was increased significantly.
并且增加了正常小鼠和垂体后叶素诱发的心肌缺血小鼠的心肌营养血流量。
Studies in the past have shown that male mice created without NPY are more anxious than normal mice, which is hardly surprising.
过去的研究已经表明，没有神经肽y的雄性小鼠比正常小鼠更容易焦虑，这并不足以令人惊奇。
These mice ended up getting sick a lot, and needed to eat much more than normal mice with intestinal bacteria in order to gain weight.
结果是，这些基因变异的老鼠不仅经常生病，而且要比正常老鼠吃更多才能长肥。
Compared with the normal mice, those with one mutant gene slept about 1.2 hours less, and mice with two mutant genes slept 2.5 hours less.
与正常的老鼠相比，携带一个变异基因的老鼠睡眠时间少1.2小时，而携带有2个变异基因的老鼠睡眠时间少2.5小时。
However, mice in which Angptl2 was deleted genetically were born normally and showed normal growth compared to genetically normal mice.
然而，先天血管生成素样蛋白2缺少的大鼠正常出生和正常生长，显示了和先天正常的大鼠一样。
When scientists genetically engineered mice to express this gene. they were able to stay awake for an extra of 1.2 hours than normal mice.
当科学家修改老鼠基因使其表达这一特性后，这种老鼠可比普通老鼠多清醒1.2小时。
What is surprising is that female mice without NPY, while still more anxious than normal mice, are less anxious than the males without NPY.
令人惊讶的是，比正常小鼠更容易焦虑的没有神经肽y的雌性小鼠，比没有神经肽y的男性更不容易焦虑。
In mice injected with human bladder, breast, lung, or melanoma cancer cells, PL inhibited tumor growth but showed no toxicity in normal mice.
注射人膀胱癌细胞、乳腺癌细胞、肺癌细胞或者黑色素瘤癌细胞的小鼠，给予PL可以抑制这些肿瘤的生长，但PL对正常小鼠未见毒性作用。
In mice injected with human bladder, breast, lung, or melanoma cancer cells, PL inhibited tumor growth but showed no toxicity in normal mice.
注射人膀胱癌细胞、乳腺癌细胞、肺癌细胞或者黑色素瘤癌细胞的小鼠，给予PL可以抑制这些肿瘤的生长，但PL对正常小鼠未见毒性作用。
应用推荐