• Recent researches confirm that the neurons containing NO synthase can be shown by NADPH-diaphorase histochemical method.

    近年研究证明,NADPH -黄递酶组化法可选择性地显示NO合成酶神经元。

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  • Adenosine stimulated a significant arteriolar nitric oxide (NO) production and dilation, which were sensitive to NO synthase inhibitor L-NMMA.

    腺苷能够显著激发动脉一氧化氮(NO)的产生而使血管舒张,且对一氧化氮合酶抑制剂L -NMMA 十分敏感。

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  • Nitric Oxide(NO) mediate a number of physiological functions in hepatocytes, and the pathological processes of viral hepatitis NO synthase(NOS).

    一氧化氮(NO)参与肝细胞的多项生理功能调节,参与病毒性肝炎的病理过程。

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  • Objective To study whether nitric oxide (NO) and neuronal NO synthase (nNOS) were involved in the pathogenesis of acute focal cerebral ischemia and reperfusion.

    目的探讨一氧化氮(NO)和神经元型NO合酶(n NOS)是否参与急性局灶性脑缺血再灌注的发病机理。

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  • The activities of organic acids-metaboling enzymes showed: There was no relation between the activity of citrate synthase(CS) and organic acids concentration.

    有机酸相关代谢酶的活性表现为:柠檬酸合酶(CS)活性变化与有机酸含量相关性不大;

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  • Objective To study effect of acupuncture on the content of nitric oxide (NO) and the activity of nitric oxide synthase (NOS) in the hippocampus of the obese rat.

    目的:探讨针刺对肥胖大鼠海马组织一氧化氮(NO)含量和一氧化氮合酶(NOS)活性的影响。

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  • Nitric oxide(NO) plays an important role in cardiovascular system. Nitric oxide synthase is the key enzyme during the synthesis process of nitric oxide.

    一氧化氮在心血管疾病中发挥了重要作用,一氧化氮是在一氧化氮合酶催化下生成的。

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  • Moreover, pre-treatment of wild type mice with a nitric oxide (. NO) synthase inhibitor prevented the HBO2-induced elevation in stem cell factor and circulating stem cells.

    而且,野生型小鼠用NO合酶抑制剂预处理后阻止了HBO2介导的干细胞因子和外周血干细胞的增加。

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  • Moreover, pre-treatment of wild type mice with a nitric oxide (. NO) synthase inhibitor prevented the HBO2-induced elevation in stem cell factor and circulating stem cells.

    而且,野生型小鼠用NO合酶抑制剂预处理后阻止了HBO2介导的干细胞因子和外周血干细胞的增加。

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