We hypothesized that alterations in cellular protein S-nitrosylation may contribute to hyperglycemia-induced endothelial dysfunction.
我们假设细胞蛋白s -亚硝基化的变化会引起高血糖介导的内皮功能障碍。
We hypothesized that alterations in cellular protein S-nitrosylation may contribute to hyperglycemia-induced endothelial dysfunction.
我们假设细胞蛋白s -亚硝基化的变化会引起高血糖介导的内皮功能障碍。
应用推荐