Objective To study whether nitric oxide (NO) and neuronal NO synthase (nNOS) were involved in the pathogenesis of acute focal cerebral ischemia and reperfusion.
目的探讨一氧化氮(NO)和神经元型NO合酶(n NOS)是否参与急性局灶性脑缺血再灌注的发病机理。
Objective To study whether nitric oxide (NO) and neuronal NO synthase (nNOS) were involved in the pathogenesis of acute focal cerebral ischemia and reperfusion.
目的探讨一氧化氮(NO)和神经元型NO合酶(n NOS)是否参与急性局灶性脑缺血再灌注的发病机理。
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