• Some physiological and psychological phenomena are connected with the dynamic behaviors of the neural networks which are constructed with this neuronal model.

    该神经元模型构造的神经网络的动力学行为可以联系到一些神经生理现象和心理现象。

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  • Base on the model we have introduced previously, we consider the effect of temperature to neuronal action potential.

    在前面介绍模型的基础上,本文考虑温度对神经元动作电位变化的影响。

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  • Objective To investigate the role of nitric oxide in neuronal damage in mice model of Parkinson's disease.

    目的研究一氧化氮在帕金森病小鼠模型神经损害中的作用。

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  • In this paper, a Markov model of finite fuzzy neuronal assembly activity is presented, and Poisson approximation is considered.

    本文给出了有限论域上模糊神经元集团活动的马尔克夫模型,并给出了泊松近似模型。

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  • Objective: To investigate the roles and relationships among ERK pathway, P53/P21 and CDK5/P35 during neuronal differentiation using a differentiative model of PC12 cells induced by NGF.

    目的:利用NGF诱导的PC12细胞分化模型,探讨ERK通路、P53/P21蛋白和CDK5/P35蛋白在神经元分化过程中的可能作用及相互关系。

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  • Now researches on the mechanism rely on the model of experimental animals and cultured neuronal cells.

    目前,动物传染性海绵状脑病的研究主要依赖于实验动物和培养神经细胞两种模型。

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  • Objective To investigate the role of nitric oxide (NO) in neuronal damage in the rat model of Parkinson, s disease (PD).

    目的研究一氧化氮(NO)在帕金森病(PD)大鼠模型神经损伤中的作用。

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  • Methods We use the model of cortical neuronal culture in neonatal rats in vitro and observe the NSE, NGF and IGFR expressions in cultured neurons by immunohistochemical method.

    方法采用体外新生大鼠皮层神经细胞培养法观察微量元素碘对神经细胞表达nse、NGF和IGFR蛋白的影响。

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  • SH-SY5Y cells are an adrenergic clone of the human neuroblastoma cell line SK-N-SH and a very useful model for the study of neuronal cell terminal differentiation.

    SY5Y细胞是人神经母细胞瘤细胞系SK-N-SH中的一个克隆,它是研究神经细胞终末分化的一个极有用的模型。

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  • Methods A model of neuronal injury was established on SH-SY5Y cells through activation of complement by cobra venom factor, an alternative pathway-specific activator.

    方法采用眼镜蛇毒因子特异激活血清补体,诱导人神经母细胞瘤细胞SH-SY5Y损伤。

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  • Methods A model of neuronal injury was established on SH-SY5Y cells through activation of complement by cobra venom factor, an alternative pathway-specific activator.

    方法采用眼镜蛇毒因子特异激活血清补体,诱导人神经母细胞瘤细胞SH-SY5Y损伤。

    youdao

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