Growth and metastasis of tumors are dependent on neovascularization.
肿瘤的生长和转移依赖于肿瘤血管新生。
To discuss the effective treatment measures of corneal neovascularization.
探讨角膜新生血管的有效治疗措施。
RESULTS: Choroidal neovascularization developed in 282 eyes of 225 patients.
结果：225名患者的282只眼睛都发生了脉络膜新生血管。
Medical treatment to inhibit retinal neovascularization will give them effective help.
药物抑制视网膜新生血管有助于对此类疾病的治疗。
Neovascularization of the muscle sarcomeres to increase blood flow through the muscles.
新生肌肉肌节血管，提高肌肉的血流量。
Vascular endothelial growth factor can induce the formation of subretinal neovascularization.
血管内皮生长因子可诱导视网膜下新生血管的形成。
The neovascularization is their mutual pathological change, and is the main reason of blindness.
新生血管形成是这些眼病共有的病理性改变，并且是其致盲的主要原因。
The image shown demonstrates the effects of a severe chemical injury with early corneal neovascularization.
如图所示为严重化学伤后早期发生的角膜新生血管形成。
Conclusions GMCSF-absence inhibits neovascularization during wound repair and leads to the delay of wound healing.
结论GMCSF基因缺失影响创面的新生血管化能力，进而影响创面的愈合。
Objective to study the inhibitory effects of nuclear transport factor 2 (NTF2) on retinal neovascularization in vitro.
目的体外实验研究核转运因子2 (NTF2)对视网膜新生血管的抑制作用。
Background: neovascularization is a new measure of treatment for ischemic heart disease and occlusive atherosclerosis.
背景：血管再生疗法是重症缺血性心脏病和闭塞性动脉硬化症的新治疗措施。
This article reviewed animal models of retinal neovascularization and researching development of medicine in this field.
文中对研究视网膜新生血管的动物模型及药物研究进展作一综述。
ConclusionCD105 may be used as a new marker of neovascularization in the research and treatment of anti-tumor angiogenesis.
结论CD 105可以作为新生血管的标记物用于抗肿瘤血管形成的研究和治疗。
Tumor cells release soluble angiogenic factors inducing neovascularization, a process referred to as the "angiogenic switch".
肿瘤细胞释放可溶性的促血管生成因子诱导新生血管生成。
Choroid neovascularization (CNV) is one of the main reasons for many ocular diseases that can lead to severe visual impairment.
脉络膜新生血管是引起多种眼病视力下降、甚至致盲的主要原因之一。
Retinal neovascularization and its hemorrhage , exudation and proliferation are major pathologic process in many ocular diseases .
视网膜新生血管的生长及其所致的出血，渗出，增生等是多种致盲眼病的主要病理过程。
When the effects of enhancing factors are stronger than those of inhibiting factors, it will cause unrestricted tumor neovascularization.
当促进因子的作用强于抑制因子时，便会引起肿瘤血管的生长。
Objective to observe the feature of age-related macular degeneration and choroidal neovascularization on optical coherence tomography (OCT).
目的应用光学相干断层扫描(oct)观察年龄相关性黄斑变性的不同特征。
Corneal neovascularization (CNV) is one of the most causes of corneal blindness and the high-risk factor of corneal graft rejective reaction.
角膜新生血管是角膜盲的主要原因，亦是角膜移植排斥反应的高危因素。
DATA SYNTHESIS: corneal neovascularization is the common pathological changes as corneal injury, inflammation and infection and many diseases.
资料综合：角膜新生血管是角膜外伤、炎症、感染等多种疾病共同的病理改变。
Conclusion: Our study did not confirm the high beneficial value of sclerosing neovascularization in patients with midportion Achilles tendinopathy.
结论：我们的研究并没有证实为跟腱中段病变的患者注射硬化剂有很多的好处。
Choroidal neovascularization is the congenerous pathologic pattern of many fundus oculi disease. It is a sign of poor sight or blind of these disease.
脉络膜新生血管是很多眼底疾病的共同病理过程，大多病例会转化为不可逆的盲，是视力预后极差的象征。
AIM: to investigate the phase change of vascular endothelial growth factor (VEGF) expression in laser-induced choroidal neovascularization (CNV) in rats.
目的：探讨血管内皮生长因子(VEGF)在激光诱导的大鼠脉络膜新生血管(CNV)生成中表达的时相变化。
Objective To investigate the clinical results of krypton red laser transpupillary thermotherapy for patients with choroidal neovascularization(CNV) at macular region.
目的探讨用氪红激光温热疗法治疗黄斑部脉络膜新生血管（CNV）的临床效果。
Tumor angiogenesis inhibitor is a class of new drugs that can destroy or inhibit tumor neovascularization, and can effectively block the growth and metastasis of cancer.
肿瘤血管生成抑制剂是一类能破坏或抑制血管生成，有效地阻止肿瘤生长和转移的药物。
Objective to observe the expression of TFPI-2 in corneal keratocytes in vitro and investigate the experimental basis for curing corneal neovascularization with gene-therapy.
目的探讨角膜基质细胞组织因子途径抑制物2 (TFPI - 2)的表达情况，为角膜新生血管的基因治疗提供方法。
Objective to observe the expression of TFPI-2 in corneal keratocytes in vitro and investigate the experimental basis for curing corneal neovascularization with gene-therapy.
目的探讨角膜基质细胞组织因子途径抑制物2 (TFPI - 2)的表达情况，为角膜新生血管的基因治疗提供方法。
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