The restenosis of vein grafts which result from neointima and atherosclerosis has became an urgent problem to be resolved.
新内膜形成和动脉粥样硬化导致的静脉桥再狭窄已成为一个亟待解决的问题。
Objective to investigate the effect of rosiglitazone on neointima and inflammatory factor after carotid artery injury of rats.
目的观察罗格列酮对大鼠颈动脉损伤后新生内膜及炎症因子的影响。
The neointima of vascular graft formed completely, but the coverage of vascular endothelial cells was incomplete in both groups.
两组动脉人工血管均通畅，内膜完整，但内皮细胞覆盖不完全。
When we use all-trans retinoic acid, neointima formation is inhibited, and the phenomenon of replicative senescences is tend to decrease.
应用血管内膜增生抑制剂后，内膜增生受到抑制，细胞增生性衰老的现象也趋于减少。
On 7th day after balloon injury showed non-uniform thickening of the neointima and mostly were made up by smooth muscle cells, vascular smooth muscle cell proliferated in the clear.
损伤后第7天，可见新生内膜非均匀增厚，多由平滑肌细胞形成；血管中膜平滑肌细胞增殖明显。
Methods Using New Zealand Rabbits, carotid atherosclerotic stenosis (CASS) model were created. The expression of ICAM-1 and MMP-2 of neointima after CEA were detected by immunohistochemical technique.
方法以新西兰兔颈动脉粥样硬化性狭窄动物模型为基础，采用免疫组织化学的方法检测颈动脉内膜切除术后不同时间点新内膜中ICAM - 1、MMP - 2基因的表达。
Methods Using New Zealand Rabbits, carotid atherosclerotic stenosis (CASS) model were created. The expression of ICAM-1 and MMP-2 of neointima after CEA were detected by immunohistochemical technique.
方法以新西兰兔颈动脉粥样硬化性狭窄动物模型为基础，采用免疫组织化学的方法检测颈动脉内膜切除术后不同时间点新内膜中ICAM - 1、MMP - 2基因的表达。
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