The University of Leeds research team has, for the first time, shown that leg muscle dysfunction is related to the severity of symptoms in heart failure patients.
英国利兹大学的研究团队首次发现腿部肌肉功能障碍与心脏衰竭患者病症严重程度之间的相关关系。
The etiology of V pattern deviation are vertical muscle dysfunction, including the primary inferior oblique muscle overreaction 28 (58%), congenital superior oblique muscle paralysis 20 (42%).
引起V型斜视的常见原因为斜肌功能异常，包括原发性下斜肌功能亢进28例（58％），先天性上斜肌麻痹20例（42％）。
Statins are known to cause muscle aches and liver dysfunction; warnings of both possibilities are prominent in the printed instructions patients get when they pick up their prescriptions.
众所周知，司他汀会造成肌肉疼痛和肝功能异常；当患者拿到自己的药方时，在印刷的用法说明书上这两种可能性的警告显而易见。
Signs of low back tenderness (58.8%), abnormal pinprick sensation (58.0%), lumbar dysfunction (43.3%), and muscle weakness of the lower limbs (42.3%) were commonly observed.
临床体征以腰背部压痛(58.8%)、下肢皮肤针刺觉异常(58.0%)、腰椎活动受限(43.3%)和下肢肌力减弱(42.3%)多见。
Conclusion in viral myocarditis, the expression of connexin 43 in the involved cardiac muscle cells was restrained, that may result in dysfunction of gap junction communication.
结论病毒性心肌炎时受累的心肌细胞连接蛋白43的表达受到抑制可能是导致心肌细胞缝隙连接通讯障碍以致心律失常的一个原因。
Residual paralysis of muscle relaxation is one of the risk factors of postoperative respiratory dysfunction, which is attracting more attention.
肌松药的残余阻滞作用是引起术后呼吸功能不全的危险因素之一，已日趋引起重视。
Conclusion in myocarditis, expression of connexin 43 and desmin in the involved cardiac muscle cells was inhibited, resulting in dysfunction of gap junctional communication and arrhythmia.
结论病毒性心肌炎时受累的心肌细胞连接蛋白43和结蛋白的表达受到抑制，可能是导致心肌细胞缝隙连接通讯和收缩功能障碍以致心律失常的一个原因。
CONCLUSION: in myocarditis, expression of connexin 43 and desmin in the involved cardiac muscle cells was inhibited, resulting in dysfunction of gap junctional communication and arrhythmia.
结论：在心肌炎，连接蛋白43和结蛋白表达的参与心肌细胞受到抑制，在间隙连接通讯功能障碍引起的心律失常。
CONCLUSION: in myocarditis, expression of connexin 43 and desmin in the involved cardiac muscle cells was inhibited, resulting in dysfunction of gap junctional communication and arrhythmia.
结论：在心肌炎，连接蛋白43和结蛋白表达的参与心肌细胞受到抑制，在间隙连接通讯功能障碍引起的心律失常。
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