Here is to review on the correlation of mitochondrial dysfunction and the pathogenesis of AS.
现就线粒体功能障碍与AS形成之间的关系进行综述。
This study overviews the researches about the diseases caused by the mitochondrial dysfunction.
本文就有关线粒体功能异常所引起的疾病的研究进展作一综述。
What they discovered was the aforementioned mitochondrial dysfunction that lead to problems with energy.
他们发现的正是前面提到的导致能量供应问题的线粒体功能异常。
Some third effect could be triggering both the symptoms of autism and the mitochondrial dysfunction.
某个第三方效应可能同时引发了自闭症和线粒体功能失调。
These are all potentially treatable and reversible causes of mitochondrial dysfunction that have been clearly documented.
这些是已经证实了的造成线粒体异常的原因，都是可以医治的。
Recently, there are more research focusing on the relationship between mitochondrial dysfunction and pathogenesis of AS.
近年来，有关线粒体功能障碍与AS关系的研究日益增多。
On the other hand, mitochondrial dysfunction can lead to many neurodegenerative conditions as well as metabolic diseases such as diabetes.
在另一方面，线粒体功能障碍可导致许多神经退行性条件，以及代谢性疾病，如糖尿病。
Objective To study enterocyte mitochondrial dysfunction of neonatal rats undergoing surgical stress and the protective effects of melatonin.
结论线粒体损伤是外科应激后新生鼠小肠上皮细胞肠道损伤的早期表现。
Mitochondrial dysfunction is measured by counting the number of mistakes they make when they transcribe their DNA code to make essential materials like proteins.
线粒体功能不良的测定，是通过计算当他们转录自身dna密码到生成必须物质，如蛋白，发生错误的数量。
Hence, it is not surprising that treatment of mitochondrial dysfunction and development of mitochondrion-targeted therapeutics have become a primary focus of scientific research.
因此，治疗线粒体功能障碍以及开发定位于线粒体的疗法已成为科学研究的首要重点不足为奇。
The decrease of mitochondrial cristal membrane density of hypoxic ischemic newborn rats was tightly related to dysfunction of mitochondrial respiration and energy synthesis.
缺氧缺血新生鼠嵴膜密度的减少与线粒体呼吸功能、能量合成功能障碍密切相关。
An underlying mechanism for endothelial dysfunction is the enhanced generation of endothelial mitochondrial reactive oxygen species (ROS).
内皮细胞功能紊乱的根本机制在于内皮细胞线粒体活性氧簇(ROS)生成过多。
Conclusion:CDCA can damage mitochondrial membrane structure and result in liver dysfunction.
结论：CDCA破坏线粒体膜和引起肝功能减退。
Conclusion:CDCA can damage mitochondrial membrane structure and result in liver dysfunction.
结论：CDCA破坏线粒体膜和引起肝功能减退。
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