Chinese Medicine, 912, may prevent rat liver mitochondria damage through reducing O 2 - and MDA.
中药912液可以提高肝细胞线粒体的抗氧化潜能，具有保护肝细胞的作用。
"Maybe the reason why free radicals and aging are correlated is because free radical production in the mitochondria (part of the cell) is a stress reaction to the damage of aging," Hekimi said.
海克米说：“人们之所以把自由基和衰老联系在一起，可能是因为自由基在线粒体(细胞的一部分)中产生是人体对衰老的应激反应。”
In recent years, evidence has been mounting that damaged mitochondria contribute to the neurological damage wrought by the disease.
近年来，有证据已经表明受到损伤的线粒体是疾病引起神经伤害的原因。
Spirulina can inhibit the above changes in myocardial mitochondria after exhaustive exercise, and then it plays a role in protecting myocardium and against exercise induced damage.
螺旋藻可抑制力竭运动后心肌线粒体的上述变化而起到保护心肌，抗运动性损伤的作用。
Over time, mitochondria accumulate damage, causing cells and eventually tissues to malfunction and break down.
随着时间的推移，线粒体的损伤逐渐积累下来，使细胞和组织产生病变，并最终被破坏。
In eggs, mitochondria have only one copy of this DNA, making them vulnerable to damage as women age.
卵细胞中线粒体的DNA只有一个副本，随着女性的年龄增长，他们极易受到伤害。
Renal interstitial fibrosis; Renal tubular epithelial cell; Mitochondria oxidative damage; Renal tubular function.
肾间质纤维化；肾小管上皮细胞；线粒体氧化损伤；肾小管功能。
This article studies the effect of spirulina (SA) and its complex prescription (SACP) protecting myocardial mitochondria in trained mice from the damage made by peroxidation.
实验研究螺旋藻及其复方对运动小鼠心肌线粒体过氧化损伤的保护作用。
Conclusion the damage of CE on the calcium pump of mitochondria might be irreversible because of the formation of covalent binding of CE-target macromolecules.
结论氯乙醇对鼠肝线粒体钙泵的不可逆抑制作用可能与共价结合有关。
Results:Observed by transmission electron microscopy:the number of mitochondria of the damp-heat in large bowel group rich, or a small number of mitochondrial cristae damage;
结果：通过投射电镜观测到：大肠湿热组线粒体较丰富，少数线粒体膜或嵴破坏；
Objective to study the morphologic changes of mitochondria and metabolic protective effect of fructose 1 6 diphosphate (FDP) intervention in newborn rats with hypoxic ischemic brain damage (HIBD).
目的探讨16二磷酸果糖(FDP)干预治疗新生鼠缺氧缺血性脑损伤(HIBD)后脑神经细胞线粒体形态学变化及对能量代谢的影响。
Objective to study the morphologic changes of mitochondria and metabolic protective effect of fructose 1 6 diphosphate (FDP) intervention in newborn rats with hypoxic ischemic brain damage (HIBD).
目的探讨16二磷酸果糖(FDP)干预治疗新生鼠缺氧缺血性脑损伤(HIBD)后脑神经细胞线粒体形态学变化及对能量代谢的影响。
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