Research on the mechanism of diabetic nephropathy was initially focused on the mesangial matrix and glomerular basement membrane.
有关糖尿病肾病发病机制的研究，人们早先关注的是肾脏系膜基质堆积和肾小球基底膜增厚。
The effect of LMWH on patients with proliferative glomerulonephritis may be associated with inhibiting the proliferation of mesangial cell and the accumulation of mesangial matrix.
提示，临床上LMWH能通过对增生性肾炎患者系膜细胞增殖和系膜基质积聚的抑制作用而发挥效应。
Objective To observe the effect of bradykinin(BK) on kidney mesangial cell multiplication and extracellular matrix secretion under high glucose environment.
目的观察缓激肽（BK）对高糖浓度下肾小球系膜细胞增殖和细胞外基质分泌的影响。
Objective To elucidate the effect of Yan-Shen Capsule on glomerular mesangial cell apoptosis and extracellular matrix metabolism of remnant kidney.
目的探讨“延肾胶囊”对残肾肾小球系膜细胞凋亡及其细胞外基质代谢的影响。
Conclusion: in vitro high glucose solution increased mesangial cell synthesis of extracellular matrix proteins.
结论：体外高糖可增加细胞外基质蛋白的合成。
Conclusion: Fenofibrate can inhibit high glucose-induced proliferation of mesangial cells, decrease synthesis of extracellular matrix, and increase degradation of extracellular matrix.
结论：非诺贝特能抑制高糖培养的系膜细胞增殖，减少胞外基质合成，增加胞外基质的降解。
Conclusion: Fenofibrate can inhibit high glucose-induced proliferation of mesangial cells, decrease synthesis of extracellular matrix, and increase degradation of extracellular matrix.
结论：非诺贝特能抑制高糖培养的系膜细胞增殖，减少胞外基质合成，增加胞外基质的降解。
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