Plasma MDA and SOD level were determined in control and model group.
分别测定模型组与对照组大鼠血浆MDA、SOD水平。
Serum NO, MDA and SOD may be responsible for impaired cognition of MPHD.
血清no、MDA、SOD可能是与认知功能密切相关的氧化应激指标。
The levels of MDA and SOD in brain and water content were also determined.
同时检测丙二醛(MDA)、SOD活性及脑组织水含量。
Methods Serum levels of NO? MDA and SOD in 30 MC cases were measured, 18 healthy subject served as normal controls.
方法检测30例急性心肌炎患儿NO、丙二醛（MDA）及超氧化物歧化酶（SOD）含量，选择18例健康体检者为对照组。
There was significant difference in the intestinal contents of MDA and SOD between B and T groups at each time point ( P <0.05).
烧伤治疗组与烧伤对照组伤后各时相点比较，差异有统计学意义（P<0。 05）。
Examine the MDA and SOD of plasma before transplantation and 1, 3, 5, 7, 9 days after transplantation for two groups, respectively.
分别检测两组受体移植前和移植后1、3、5、7、9天的血浆丙二醛含量和超氧化物歧化酶活性。
To explore the effects of L-arginine on the brains of fetal rats with IUGR and the relationship with the levels of NO, MDA and SOD.
目的了解左旋精氨酸对被动吸烟所致大鼠宫内发育迟缓及胎鼠脑发育的影响，通过其与NO、MDA和SOD的关系探讨其作用机制。
The parameters of blood levels of MDA and SOD in rats were observed. The rate of apoptosis adrenal gland and pituitary tissues of rats were examined.
以大鼠血液SOD活性水平和MDA含量以及肾上腺和脑垂体组织的细胞凋亡率作为检测指标进行检测。
The effects of baicalin on ischemia-induced barriers to behavior, rate of infarction, water content of brain tissue, contents of NO, NOS, MDA and SOD in brain tissues were observed.
结果黄芩苷可以明显改善大鼠脑缺血再灌注所致的行为学障碍，降低梗死率，降低脑组织含水量，同时可以降低脑内NO、NOS和MDA的含量，增加SOD含量。
Then we examine their serum malondialdehyde (MDA) and superoxide dismutase (SOD), compare the difference of MDA and SOD every group, analysis the correlation of MDA and SOD with BMD.
检测各组研究对象的血清丙二醛（MDA）、超氧化物歧化酶（SOD），比较各组之间的MDA、SOD的不同，分析MDA、SOD与BMD的相关性。
Objective To study the influence of oral magnetic field treated spirits for superoxide dismutase(SOD)activity, malonyldialdehyde(MDA)and nitric oxide(NO)level in human body.
目的研究饮用磁处理白酒对人血清超氧化物歧化酶（SOD）活性、丙二醛（MDA）及一氧化氮（NO）含量的影响。
The effects of SOD and POD activity and MDA amount are different between different treatments.
不同处理的不同浓度对保护酶活性和MDA含量影响不同。
And can improve SOD vigor in the blood and reduce MDA content, have better oxidation of resisting.
能提高血液中SOD活力和降低MDA含量，具有较好的抗氧化作用。
Objective:To probe the changes of SOD, MDA and NO in the brains of VD mice by bovine acidic neuropeptide-1(BANP-1).
目的：探讨酸性神经肽1（BANP-1）对血管性痴呆（VD）小白鼠脑内超氧化物歧化酶（SOD）、丙二醛（MDA）和一氧化氮（NO）的影响。
The results showed that hyperin could increase SOD activity and NO amount of the tissues and decrease MDA level, suggested that hyperin may play its pharmacological role through above mechanisms.
结果表明：金丝桃甙能增加组织SOD活性及NO含量，降低MDA水平，可能是其发挥药理作用的机制之一。
Ciwujia capsule can protect the colon tissue of depressed rats by changing the levels of MDA, SOD and the expression of COX 2.
刺五加胶囊可能通过改变抑郁模型大鼠结肠组织mda,SOD和COX2的表达来改善其结肠的功能。
The effect of fonr trace elements, malonaldehyde(MDA )and superoxide dismutase (SOD) activity were observed with with. self-control method.
用自身对照方法，观察服灵芝前后铅、锌、铜、锰、铁、丙二醛和过氧歧化酶（SOD）活性的改变。
Objective: To explore the correlation of degenerative osteoarthritis and NO, SOD, MDA in serum.
目的：探讨退行性膝关节病与血清中NO、SOD、MDA相关性。
Superoxide Dismutase (SOD) and Malondialdehyde (MDA) were measured to show the degree of lipid peroxidation.
以超氧化物岐化酶(SOD)活性和丙二醛(MDA)的含量反映脂质过氧化程度。
The content of SOD and MDA in synovium was detected after treatment.
检测治疗后滑膜组织中SOD和MDA的含量。
Determine cerebral homogenate SOD, MDA and cerebral humidity content.
测定脑匀浆sod、MDA含量和脑组织含水量。
Plasma lipid, malodiadehyde (MDA) and superoxide dismutase (SOD) were assayed according to kit manu.
同时测定血脂、丙二醛（MDA）和超氧化物歧化酶（SOD）。
The tissues and blood were obtained, in which the vitality of SOD and the content of MDA were detected.
处死小鼠后取肝、脾、肾组织及血清，测定超氧化物歧化酶（SOD）活力及丙二醛（MDA）含量。
Conclusion: During chronic lead poisoning, lead poisoning results in the decrease of SOD activity and the increase of MDA content as time goes by.
结论：慢性染铅过程中，肾脏sod活性随染铅时间延长逐渐降低，MDA含量随染铅时间延长逐渐增加。
The concentration of malondialdehyde(MDA), superoxide dismutase (SOD) and myeloperoxidase (MPO) in plasma and in liver, kidney and intestine were determined at different times in all groups.
观察血浆丙二醛（MDA）、超氧化物歧化酶（SOD）及组织中丙二醛和髓过氧化酶（MPO）的含量变化，并观察小肠、肝、肺的病理变化。
Methods SOD activity and MDA content in rats 'tissues among control (c) group, diabetic (d) group and insulin therapy (ID) group were determined.
方法测定对照组(C)、胰岛素治疗糖尿病组(ID)、糖尿病组(D)大鼠重要器官组织sod活力、MDA含量。
The changes of neurological defect were observed. The content of brain water, MDA, NO and the activity of SOD were measured.
观察神经缺损症状的变化及检测脑系数、脑含水量、丙二醛(MDA)含量、超氧化物歧化酶(SOD)活性、一氧化氮(NO)含量。
Through establishing mice model experiments, the effects of APFM on MDA content and superoxide dismutase (SOD) in the organs of diabetic mice induced by alloxan were observed and analyzed.
在小鼠模型实验中，观察和分析APFM对四氧嘧啶所致糖尿病模型小鼠脏器中MDA含量和SOD活性等生理指标的影响。
Through establishing mice model experiments, the effects of APFM on MDA content and superoxide dismutase (SOD) in the organs of diabetic mice induced by alloxan were observed and analyzed.
在小鼠模型实验中，观察和分析APFM对四氧嘧啶所致糖尿病模型小鼠脏器中MDA含量和SOD活性等生理指标的影响。
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