Conclusion LP-PLA2 is the danger factor of CHD, it contribute to CHD through damaging the endothelial cell function mechanism.

结论：LP - PLA2是CHD发病的危险因素，它可能通过损伤内皮细胞的功能增加患病风险。

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Objective:To investigate the expression of Lp-PLA2 and LPA in rabbit's cerebral ischemia and their correlationship.

目的：研究大鼠脑缺血损伤后，脂蛋白磷脂酶A2（Lp-PLA2）与溶血磷脂酸（LPA）的表达及相互关系。

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Methods:The rats' models of sodium urate arthritis were mode, and the effects of gouty granule on LP-PLA2 value and swollen right ankle joint degrees of rat model were observed.

方法复制大鼠尿酸钠关节炎模型，观察痛风颗粒对大鼠右踝关节肿胀度、LP-PLA2值的影响。

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Results: Gouty granule inhibited the ankle swelling of rats' models obviously, and reduced serum value of LP-PLA2 significantly.

结果痛风颗粒对大鼠踝关节肿胀有明显的抑制作用，能显著降低大鼠血清lp - PLA2值，与秋水仙碱作用相近。

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Results: Gouty granule inhibited the ankle swelling of rats' models obviously, and reduced serum value of LP-PLA2 significantly.

结果痛风颗粒对大鼠踝关节肿胀有明显的抑制作用，能显著降低大鼠血清lp - PLA2值，与秋水仙碱作用相近。

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