The boundary of liver lobule and pancreatic lobule was obscure.
肝脏和胰脏的小叶界限不清晰。
After day 18, the normal liver lobule structures began to recover.
第18天以后，正常的肝小叶结构开始恢复。
As the liver increasing and the liver lobule forming, 1-2 layers of cells arrange in the liver cell cords.
随着肝脏增大，肝小叶的形成，排列成由1 ~2层甚至单层的肝细胞索。
In the control group, the structure of liver lobule was normal and a few of collagen fibers located in portal area and central vein.
对照组肝小叶结构和肝细胞状态正常，仅在中央静脉及汇管区可见少量胶原纤维。
Fatty liver disease (FLD) is a clinicopathological syndrome caused by a variety of reasons. The main lesions are in hepatic lobule with steatohepatitis and accumulation of triglycerides in the liver.
脂肪性肝病是多种原因引起的病变主体在肝小叶、以肝细胞脂肪变性和脂肪蓄积过多为主的临床病理综合征。
Results 12 weeks after carbon tetrachloride administration, the rabbit liver developed typical pseudo-lobule, and the liver hydroxyproline content increased 3.5 times compared to normal control.
结果四氯化碳注射12周后，兔肝组织出现典型的假小叶，肝组织羟脯氨酸含量较正常组升高3.5倍。
Results 12 weeks after carbon tetrachloride administration, the rabbit liver developed typical pseudo-lobule, and the liver hydroxyproline content increased 3.5 times compared to normal control.
结果四氯化碳注射12周后，兔肝组织出现典型的假小叶，肝组织羟脯氨酸含量较正常组升高3.5倍。
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