Objective To investigate the affinity of lipid-coated microbubbles (LCMs) for an injured site in rat brain, and discuss the mechanism of LCM targeting brain lesions.
目的研究脂膜微囊向脑损伤位点靶向性聚集的特点，探讨其向鼠脑损伤区靶向性聚集的机制。
Conclusions Lipid-coated microbubbles may carry Taxol to C6 glioma cells, and the compound may act an killing effect in vitro. The mechanisms of metabolism associated with acidic component.
结论脂膜微囊可以承载紫杉醇在体外起到杀伤胶质瘤细胞的作用，且脂膜微囊的代谢与细胞内的酸性成分有关。
Conclusions Lipid-coated microbubbles may carry Taxol to C6 glioma cells, and the compound may act an killing effect in vitro. The mechanisms of metabolism associated with acidic component.
结论脂膜微囊可以承载紫杉醇在体外起到杀伤胶质瘤细胞的作用，且脂膜微囊的代谢与细胞内的酸性成分有关。
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