The research of overloading on cellular level may elucidate the mechanical effect on the formation, regeneration and growth of bone and the mechanism of cell response in bone.
细胞水平过载的研究可使人们进一步搞清应力对骨形成、重建和生长的影响及骨内细胞响应机制。
Results Dexamethasone beyond physical dosage could suppress cells proliferation, reduce SOD level, increase MDA, and cause cells injury and Ca 2+ overloading.
结果超生理剂量地塞米松可抑制骨骼肌细胞的增殖，降低其SOD水平，提高MDA水平，并造成细胞的损伤与钙超载。
Results Dexamethasone beyond physical dosage could suppress cells proliferation, reduce SOD level, increase MDA, and cause cells injury and Ca 2+ overloading.
结果超生理剂量地塞米松可抑制骨骼肌细胞的增殖，降低其SOD水平，提高MDA水平，并造成细胞的损伤与钙超载。
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