Compared with the normal mice, those with one mutant gene slept about 1.2 hours less, and mice with two mutant genes slept 2.5 hours less.
与正常的老鼠相比，携带一个变异基因的老鼠睡眠时间少1.2小时，而携带有2个变异基因的老鼠睡眠时间少2.5小时。
Again, the mutant mice behaved appropriately, but they were much less aggressive, spent less time fighting and they took longer breaks between attacks.
这个基因改变的老鼠表现是适当地，可是它们的攻击性减小、打斗的时间也减少而且在打斗之间的休息时间更长。
And Susheng Gan, a molecular biologist at Cornell University, says the mutant type used by the group shows only slightly delayed leaf senescence, making it a less-than-ideal model to work with.
甘肃升sushengGan是卡奈尔大学的分子生物学家，说改组使用的变异物种只稍微推迟了树叶的衰老表象，这不是很理想的模型。
However, mutant ABCG2 can be less efficient at pushing gefitinib out of cells in the intestine, the researchers noted. A buildup of drug within these cells can cause diarrhea.
研究人员指出，突变的ABCG2降低了肠道细胞“泵出”的功能，使得细胞内的药物逐渐累积从而导致腹泻。
But the researchers found that such remodeling was impaired in the mutant mice that produced less nitric oxide when compared to their normal counterparts.
但研究人员发现，这种重构是在遗传突变小鼠中由于一氧化氮减少而受损，这是与正常小鼠相比较的结果。
Some people even have a genetic defect in the enzymes in their liver that break down nicotine, whereby the mutant enzyme is much less effective at metabolizing nicotine than the normal variant.
有些人的肝脏中分解尼古丁的酶有遗传上的缺陷，因此这种突变的酶在代谢尼古丁时的作用就比正常的变异体的作用小。
Some people even have a genetic defect in the enzymes in their liver that break down nicotine, whereby the mutant enzyme is much less effective at metabolizing nicotine than the normal variant.
有些人的肝脏中分解尼古丁的酶有遗传上的缺陷，因此这种突变的酶在代谢尼古丁时的作用就比正常的变异体的作用小。
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