Karpas-299 and K562 cells were lysed without starvation or treatment.
Karpas- 299和K562细胞在裂解前未经饥饿和其它处理。
Besides, the survival cell rate of K562 was higher than that of HL-60.
K562细胞存活分数高于HL - 60细胞，与临床治疗结果相一致。
Result: Tricholoma Matsutake markedly inhibited K562 cell proliferation.
结果：松茸对K562细胞增殖具有明显的抑制作用。
Objective: To study the antitumor effect of Tricholoma Matsutake on K562 cells.
前言：目的：研究松茸多糖对K562细胞增殖与分化的影响。
Objective: to study the mechanism of apoptosis induced by Orindonin in K562 cells.
目的：研究冬凌草甲素诱导白血病k5 6 2细胞凋亡的机制。
Conclusion: ERK negatively regulated the apoptosis induced by bufalin in K562 cells.
结论：ERK负向调节蟾蜍灵诱导的K562细胞凋亡过程。
RESULTS: Microcapsules effectively protected K562 cells against the killing by NK cells.
结果：微囊可有效保护囊内k562细胞不受NK细胞的杀伤。
Objective: To study the effects of mustine and its isomeride on K562 and L1210 cell lines.
前言： 目的：研究盐酸氮芥、 氮芥异构体对K_（562）及L_（1 210）细胞增殖的影响。
The kill effects of SMF cooperated with Chinese herbs on K562 cells were tested by MTT test.
用MTT法检测磁场结合中药（土贝母皂甙、白屈菜红碱和血根碱）对K562细胞的杀伤作用。
AIM To study the proliferation of leukemia cell line K562 inhibited by glucosamine sulphate.
目的研究氨基葡萄糖硫酸盐对白血病细胞K 5 6 2增殖的影响，并探讨分子机制。
Objective To study the effect and mechanism of anti-VEGF antibody on apoptosis of K562 cells.
目的观察抗VEGF抗体对K562细胞凋亡的影响，探讨VEGF抑制白血病细胞凋亡的机理。
To study the possibility and the mechanism of K562 apoptosis induced by microwave and etoposide.
探讨微波和足叶乙苷体外诱导K562细胞凋亡的可能性及其机制。
The effect of inducers on leukemia cell line K562 was studied in liquid culture system in vitro.
本文以人白血病细胞株k _(562)为实验模型，通过体外液体培养，研究了白血病细胞诱导剂的作用。
This study was aimed to investigate the effect of D-limonene on K562 leukemia cells and its mechanism.
本研究探讨d -柠檬烯对K562白血病细胞增殖的影响及机制。
Conclusion Prazosin can inhibit the proliferation of K562 cells and induce the apoptosis of K562 cells.
结论：哌唑嗪能抑制K562白血病细胞的增殖，并能诱导其凋亡。
HL 60 and K562 cells showed typical apoptosis features of "ladder" and the sub G1 peak in DNA histogram.
作用HL60和K562细胞后电泳显示有凋亡细胞特有的“梯状”条带，DNA直方图出现亚g1峰。
Results showed that ABCG2 expression increased megakaryocytic differentiation of K562 cells induced by PMA.
结果显示，ABCG2的表达促进了PMA诱导的K562向巨核系的分化。
The research showed that the complexes can inhibit K562 tumor cell's growth, generation and induce apoptosis.
此外，还对部分配合物进行体外抗癌活性测试研究，实验靶细胞是人慢性粒细胞性白血病K562细胞。
CONCLUSION: PMA and BCR-ABL chimeric gene can influence the expression of EDAG gene in leukemia cell line K562.
结论：诱导剂佛波酯和BCR - ABL嵌合基因可以影响EDAG基因在白血病细胞系k562中的表达。
OBJECTIVE To study the mechanism of inhibition of K562 leukemia cells proliferation by glycyrrhetinic acid (GA).
目的：探讨甘草次酸(GA)抑制K562细胞增殖的机制。
The results showed that deguelin inhibited the proliferation of K562 cells in a time- and dose-dependent manner;
结果表明：鱼藤素能显著抑制K562细胞的增殖，并与作用时间和鱼藤素浓度呈依赖关系；
Objective To investigate the toxicological effects of the crude venom of Bungarus multicinctus on K562 tumor cells.
目的探讨银环蛇毒素及其若干组分在体外对白血病K5 6 2细胞株的毒性作用。
METHODS: The expression of TRAIL on K562 cells was detected by indirect immunofluorescence staining and flow cytometry.
方法：采用间接免疫荧光流式细胞术，分析K562细胞表面TRAIL分子的表达。
AIM To study the differentiation effect of leukemia cell line K562 induced by glucosamine and glucosamine hydrochloride.
目的研究氨基葡萄糖及其盐酸盐对白血病细胞K 5 6 2的诱导分化效果。
CONCLUSION: Extract sago cycas leaves has remarkable inhibiting effect on proliferation of leukemia K562 and HL-60 cells.
结论：铁树叶提取液对白血病细胞株k562及HL - 60细胞的增殖有明显抑制作用。
MPO was positive in HL60 cells. K562 cells morphology obviously showed a primitive and no-differentiation characteristic.
细胞形态学上K562细胞原始未分化特性比较明显，HL60细胞质内已有嗜天青颗粒和空泡等细胞分化特征。
The effect of Ginsenosides on the induction of differentiation in the human erythroleukemia cell line K562 was investigated.
本文以K562人类红白血病细胞系为实验模型，研究了人参皂甙对该细胞系的诱导分化作用。
Flow cytometric analysis of K562 cells using c-Rel antibody (blue) compared to a nonspecific negative control antibody (red).
流式细胞仪研究K562细胞，所用抗体为c - RelAntibody(蓝色)与非特异性的阴性对照抗体(红色)。
Objective to investigate the proliferation inhibition and apoptosis-inducing effect of Artesunate on human leukemia K562 cells.
目的观察青蒿琥酯对人白血病细胞株k562抑制增殖和凋亡作用。
Conclusions: the change of protein tyrosine kinase activity is involved in the differentiation of K562 cells induced by matrine.
结论：在苦参碱诱导K562细胞分化的过程中，涉及到蛋白酪氨酸激酶的活性改变。
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