Objective: To explore the mechanism of JNK pathway regulating proliferation of hepatic stellate cells(HSC) stimulated by acetaldehyde.
目的：初步探讨JNK信号通路调控乙醛刺激的大鼠肝星状细胞（HSC）增殖的分子机制。
CONCLUSION: Acetaldehyde causes oxidative damage in cardiomyocytes through enhancing cellular ROS level, and induces cardiocytes apoptosis by activating ROS mediated JNK pathway.
结论：乙醛通过增高胞内ros水平损伤心肌细胞，激活ros介导JNK途径诱导细胞凋亡。
Conclusion The activation of JNK signaling pathway may play an important role in t lymphocyte proliferation.
结论JNK信号通路的活化在小鼠T细胞增殖中可能起着重要作用。
Conclusion: It is suggested that JNK has effects on proliferation of HSC stimulated by acetaldehyde and JNK maybe an important signal pathway regulating HSC proliferation.
结论：JNK活性变化与乙醛刺激的大鼠hsc的增殖具有相关性，提示JNK可能是调节hsc增殖的重要信号通路之一。
JNK signaling transduction pathway plays a vivtal role in the neuronal apoptosis.
JNK信号转导途径参与神经元凋亡。
JNK signaling transduction pathway plays a vivtal role in the neuronal apoptosis.
JNK信号转导途径参与神经元凋亡。
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