Conclusion Captopril is able to preserve ischemic myocardium.
结论卡托普利对缺血心肌具有保护作用。
Reperfusion of ischemic myocardium is necessary to salvage tissue from ultimate death.
心肌缺血后的再灌注对于挽救心肌细胞死亡是非常必要的 。
It has apparently protective effects on the reperfusion injuries of ischemic myocardium.
对心肌缺血与再灌注损伤具有明显的保护作用。
Objective To investigate the protective effects of selenium on acute ischemic myocardium.
目的探讨硒对急性缺血心肌保护作用的机制。
The uptake of insulin ischemic myocardium was almost 2 times that of ischemic myocardium.
同时伴随葡萄糖摄取量明显增加，是单纯缺血心肌葡萄糖摄取量的2倍。
Conclusion MVG is more sensitive than myocardial velocity for detecting ischemic myocardium.
结论MVG是比心肌运动速度更敏感的反映局部心肌缺血的指标。
OBJECTIVE To evaluate the effects of modified cold cardioplegia on warm ischemic myocardium.
目的研究改良心肌保护液对短暂常温热缺血供心移植后的保护作用。
AIM: To observe the effect of Baoxin capsules on hemodynamics of ischemic myocardium of dogs.
目的：观察保心胶囊对结扎犬心肌缺血时心脏血流动力学的影响。
Objective: To study the effect of adenine sulphate preconditioning(ASPC) on apoptosis of rats ischemic myocardium.
目的 ：探讨硫酸腺嘌呤预处理对大鼠缺血心肌细胞凋亡的保护。
AIM: To detect the effect of Sini decoction on glutathione S-transferase (GST) mRNA expression in the ischemic myocardium.
目的：探讨四逆汤对缺血心肌谷胱甘肽s转移酶(GST)基因表达的影响。
The sensitivity and specificity of SRI with DSE for detecting ischemic myocardium were (90.0) % and (91.7) %, respectively.
应变率检测缺血节段的敏感性和特异性分别为90.0%和91.7%。
Promoting revascularization in ischemic myocardium has been a novel therapeutic strategy for ischemic cardiovascular diseases.
促进缺血心肌血管新生已成为治疗缺血性心血管疾病的新策略。
Aim: To observe the effect of LPC on the pacemaker current if in ischemic myocardium and if the effect could be reversed by ISO.
目的：观察在缺血条件下，溶血磷脂酰胆碱对心肌起搏离子流的影响以及能否被异丙肾上腺素逆转。
To investigate if insulin stimulates the translocation of glucose transporter 1(GLUT1) and glucose uptake in ischemic myocardium.
观察胰岛素能否刺激缺血心肌葡萄糖转运子1（GLUT1）移位和葡萄糖摄取。
Objective To evaluate the pharmaceutical effect of angiotensin-converting enzyme inhibitor(ACEI) captopril on ischemic myocardium.
目的评价血管紧张素转换酶抑制剂（ACEI）卡托普利对缺血心肌的药理作用。
The changes of ultrastructure of ischemic myocardium were observed with electrical microscope at 1,2,3 months after the operation.
分别在术后1、2、3个月时采用透射电镜观察缺血心肌细胞超微结构的改变情况。
The results prove that ferulic acid sodium plays an important role in resisting the reperfusion injury of the ischemic myocardium.
结果初步提示：阿魏酸钠具有抗缺血心肌再灌注损伤的作用。
Results Peak systolic strain of ischemic myocardium in patient with CHD was significantly lower, disappeared, or even showed inversion.
结果冠心病病人缺血心肌的收缩期峰值应变表现为明显减低、消失，甚至倒置。
CONCLUSION :Sini decoction can promote the expression of GST gene, which may be related to its protective effect on ischemic myocardium.
结论：四逆汤能诱导GST基因的表达，GST能催化谷胱甘肽与亲电子物质发生结合反应。
Conclusion: Baoxin Capsules can effectively decrease blood viscosity, improve circulate of coronary artery, and protect ischemic myocardium.
结论：保心胶囊可有效降低血液粘度，改善冠脉循环，保护缺血心肌。
Objective To observe the protective effects of Shengmai inject on on ischemic myocardium in patients with acute myocardial infarction (AMI).
目的：探讨生脉注射液对急性心肌梗塞(ami)缺血心肌的保护作用。
Objective To observe the influence of autologous skeletal muscle stem cell implantation into the ischemic myocardium on myocardial structure.
目的观察自体骨骼肌干细胞移植于缺血心肌后对心肌结构变化的影响。
The possible mechanism is that blood can provide energy substrate for ischemic myocardium and maintain the cellular metabolism and homeostasis.
说明血液灌注液比晶体液的保护效果明显。可能是血液在心肌缺血状态提供氧基质，保证细胞代谢和维持内环境的稳定。
Conclusion Transmyocardial laser revascularization (TMLR) can reduce the injuries of ischemic myocardium when canine coronary artery is ligated.
实验结果提示激光心肌管道的形成能减轻结扎犬冠状动脉引起的急性心肌缺血性损伤。
Objective: To investigate whether insulin stimulates the translocation of glucose transporter 4 (GLUT4) and glucose uptake in ischemic myocardium.
目的：观察胰岛素能否刺激缺血心肌葡萄糖转运子4 (GL UT4)移位和葡萄糖摄取。
CONCLUSION: the results suggested that it was one of the most important mechanisms through raising the NO levels to protect ischemic myocardium in ECP.
结论：体外反搏促进NO的产生可能是其抗心肌缺血性损伤的重要机制之一。
Conclusions PW DTI combined with high dose DSE can correctly evaluate left ventricular regional systolic function of long axis and detect ischemic myocardium.
结论PWDT I能准确评价多巴酚丁胺负荷试验中左室长轴局部心肌收缩功能，从而准确无创检测冠心病缺血心肌。
Activity of normal myocardium declined further by 63% from 30 to 240 min of reperfusion, while activity of transciently ischemic myocardium decreased by 15.5%.
经过30-240分钟的反复灌注，正常心肌活力降低了63％，而短暂性心肌缺血的心肌活力降低了15.5％。
Aim To investigate the effect of fibroblast growth factor 2 (FGF2) cDNA mediated by recombinant adeno-associated viral vector2 (rAAV-2) on ischemic myocardium.
探讨重组2型腺相关病毒载体介导2型成纤维细胞生长因子基因诱导家兔缺血心肌血管生成的作用。
The low survival rate of stem cells in the ischemic myocardium microenvironment is the bottle neck in treating ischemic diseases with stem cell transplantation.
移植干细胞在缺血心肌微环境中的低存活率是影响干细胞移植治疗效果的关键。
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