Objective: To study the neuroprotective mechanism of aspirin preconditioning in ischemic cerebral injury and its optimal dose.
目的：探讨阿司匹林预处理对缺血性脑损伤的保护机制及最佳剂量。
The pathophysiological mechanisms of ischemic cerebral injury involved in the release of excitatory amino acids, depolarization about the infarction, inflammatory reaction and neuron apoptosis.
缺血性脑损伤的病理生理机制包括兴奋性氨基酸的释放、梗死灶周围去极化、炎症反应及神经元凋亡等。
Conclusions: Aspirin preconditioning has neuroprotective effects for the subsequent ischemic cerebral injury. Its mechanism may be the inhibition of NOS activity and the reduction of NO release.
结论：阿司匹林预处理对随后的缺血性脑损伤具有保护作用，其机制可能是抑制NOS活性，减少NO释放。
CONCLUSIONS: SA reduced focal cerebral ischemic injury induced by middle cerebral artery thrombosis.
结论：SA可减轻大脑中动脉血栓所致局部脑缺血性损伤。
Study on the cytokines and their antagonists provides a new method for the therapy of cerebral ischemic injury in the future.
因此细胞因子及其拮抗剂的研究有可能为脑缺血损伤的治疗提供一种新的思路和手段。
Intercellular adhesion molecules 1 (ICAM-1) and vascular cell adhesion molecule 1 (VCAM-1) play important roles in the inflammatory process of cerebral ischemic injury.
细胞间黏附分子-1 (ICAM - 1)和血管细胞黏附分子- 1 (VCAM - 1)在脑缺血损伤炎症过程中起着重要作用。
Aim: To study the correlation between the cerebral ischemic injury and the microemboli of different composition and size.
目的：探讨微栓子不同组成和大小与脑缺血损伤程度的相关性。
The results suggested that the increased endothelin level of ischemia brain territory was important factor leading to ischemic neuronal injury, and FNZ had protective effects on cerebral ischemia.
上述结果提示：缺血脑组织内皮素含量升高是导致缺血神经元损伤的重要因素，氟桂嗪对脑缺血有保护作用。
Objective to explore the expression of growth associated protein-43 (GAP-43) to the change of plasticity of neurons axon regeneration after cerebral ischemic reperfusion injury in rats.
目的探讨脑缺血再灌注损伤后生长相关蛋白- 43 (GAP - 43)的表达对神经元轴突再生的可塑性变化。
OBJECTIVE: to study the protection of compound ferulic acid against cerebral ischemic reperfusion injury in experimental rats and to explore its function mechanism.
目的：研究复方阿魏酸对实验性大鼠脑缺血再灌注损伤的保护作用，并探讨其作用机制。
Objective To investigate the protection of brief focal cerebral ischemic preconditioning against microvascular injury and brain edema after subsequent severe focal ischemia.
目的研究短暂局灶性脑缺血预处理对再次脑缺血后微血管损伤及脑水肿的保护作用。
CONCLUSION: the acute focal cerebral ischemic injury leads to the abnormal leukocyte rheology and declining of deformability.
结论：急性局灶性脑缺血损伤中有白细胞流变学异常，变形能力下降。
OBJECTIVE: To observe the effect of chlorimipramine on motor function of rats with focal cerebral ischemic injury.
目的：观察氯丙米嗪对局灶性脑缺血损伤后大鼠运动功能的作用。
Phenytoin sodium may protect brain tissues from cerebral ischemic injury by inhibiting formation of free radical, anti-lipid peroxidation and improving regional cerebral blood flow.
苯妥英钠对缺血性脑损伤的神经保护作用与其能抑制自由基形成，减轻自由基介导的毒性，抗脂质过氧化反应以及扩张血管，增加局部脑血流量有关。
AIM To investigate the therapeutic time window for Shenfu solution in transient focal cerebral ischemic injury in rats.
目的探讨参附注射液对局灶性脑缺血性损伤的治疗时间窗。
It can be tested that cerebral ischemic injury is more and more serious with ischemic time prolonging.
说明随着缺血时间的延长，缺血对脑组织造成的损害是进行性加重的。
Conclusions:The Shenfu injection possesses dosageindependent protective effects on transient focal cerebral ischemic injury.
结论：参附注射液对大鼠短暂性局灶性脑缺血损伤呈非剂量依赖性保护作用。
Objective: To research the protective effect of pueraria flavonoid on the cerebral ischemic reperfusion injury.
目的：探讨葛根黄酮对脑缺血再灌注损伤的保护作用。
The inflammatory immune response plays an important part in cerebral ischemic injury.
免疫炎症反应在脑缺血后损伤中起重要作用。
OBJECTIVE:To investigate the protective effects of NIF, NHH, TNHH on focal cerebral ischemic injury induced by middle cerebral artery occlusion in rats.
目的：探讨基因工程药物NIF、NHH、TNHH对大鼠动脉阻塞性脑损伤的治疗作用。
Objective to observe the protective effect of total flavone of Camellia (TFC) against cerebral ischemic injury.
目的研究山茶花总黄酮(TFC)对缺血性脑损伤的保护作用。
To study the time-response relationship of isoflurane preconditioning on focal cerebral ischemic injury.
研究异氟醚预处理脑保护作用的时间效应关系。
Background How to reduce cerebral ischemic injury remain a thorny problem, also an urgent and unresolved issue.
背景如何减轻脑缺血损伤仍是一个世界性的难题和亟待解决的问题。
OBJECTIVE: To explore the protection of sodium ferulate allied with IP in cerebral ischemic-reperfusion injury.
目的：探讨阿魏酸钠联合缺血预处理对脑缺血再灌注损伤的保护作用。
The article introduces the recent effects of cytokines in the processes of cerebral ischemic injury and the survey of research of therapeutic methods. It can provide references for the deve...
文章就近年来在脑缺血损伤过程中细胞因子的作用及有关治疗方法的研究概况作了介绍，以为研制治疗脑缺血的药物提供参考。
AIM To evaluate the changes of serum and brain tissue endoxin in model of bilateral cerebral hemisphere ischemic reperfusion injury, and effect of anti digoxin antiserum (an antagonist of endoxin).
目的观察脑缺血再灌注损伤时血清和脑组织内洋地黄素水平变化和内洋地黄素拮抗剂地高辛抗血清对脑缺血再灌注损伤的干预作用。
Conclusion CXJC has protective effects on cerebral ischemic-reperfusion injury in rats.
结论CXJC对脑缺血再灌注损伤具有保护作用。
Objective:To investigate the influence of Ditan decoction on apoptosis of cerebral ischemic-reperfusion injury in rats.
目的探讨涤痰汤对大鼠局灶性脑缺血再灌注损伤细胞凋亡的影响。
Objective:To investigate the influence of Ditan decoction on apoptosis of cerebral ischemic-reperfusion injury in rats.
目的探讨涤痰汤对大鼠局灶性脑缺血再灌注损伤细胞凋亡的影响。
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