A third hypothesis suggests that chronic long-lasting mild cerebrovascular damage, including inflammatory processes and oxidative stress, may cause Alzheimer's disease.
第三个假设认为，长期的慢性轻微脑血管损伤，例如发炎和氧化压力(oxidative stress)，可能会导致老年痴呆症。
We argue that anemia of chronic disease is an adaptive response and could be beneficial to patients with inflammatory disease. Multiple lines of evidence support this hypothesis.
再来看作者的结论，已明确作者主旨：本文中作者们认为慢性病性贫血是一种适应性反应，并对慢性炎症患者有利，很多证据支持本论点。
It is assumed that VC Fat reflects post-inflammatory change but there have been no prospective studies that have actually tested this hypothesis.
VC脂肪病变可能是一种炎症后改变，暂无前瞻性研究对这种假说加以证实。
This review summarizes the possible mechanisms which include weight loss, gastrointestinal hormones, foregut hypothesis, hindgut hypothesis, adipocytokines, and inflammatory factors.
本文总结了体重减轻、胃肠激素、前肠学说、后肠学说、脂肪细胞因子和炎症因子等方面机制的研究进展。
This review summarizes the possible mechanisms which include weight loss, gastrointestinal hormones, foregut hypothesis, hindgut hypothesis, adipocytokines, and inflammatory factors.
本文总结了体重减轻、胃肠激素、前肠学说、后肠学说、脂肪细胞因子和炎症因子等方面机制的研究进展。
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