These results open a new avenue in clinical trial research in HCC.
这些结果为HCC的临床试验研究开辟了一条新的途径。
Conclusions coagulation and anticoagulation were activated in HCC.
结论肝细胞癌病人存在凝血，纤溶激活状态。
Conclusion: TCM could lessen postoperative fever in HCC patients after TACE.
结论：中药可以减轻肝癌患者介入术后出现的发热症状。
DNA methylation may be an important mechanism of inactivation of RUNX3 gene in HCC.
甲基化机制可能是肝细胞癌runx3基因失活的重要机制之一。
Conclusion Non-specific immune treatment can improve the immune function in HCC after RFA.
结论非特异性免疫治疗可提高射频消融后肝细胞癌患者的免疫功能。
Conclusion: There is decline of HLA-ABC antigen expression in HCC tissues and HCC cell lines.
结论肝癌组织和肝癌细胞系中HLA - ABC抗原表达有所下降。
Objective: To summarize the diagnosis and treatment of spontaneous rupture and hemorrhage in HCC.
目的：总结原发性肝癌自发破裂出血的诊断和治疗经验。
This study was to investigate the expression and clinical value of VEGF and MMP-9 in HCC patients.
本研究旨在探讨VEGF与MMP - 9在肝癌组织中的表达及其预测肝癌患者预后的意义。
Further clinical trials using biologic agents are warranted to prolong the survival in HCC patients.
更进一步，使用生物因子的临床实验证实能延长肝癌患者的生存时间。
Conclusion Primary resistance existed in HCC and there were many mechanisms of multidrug resistance.
结论肝细胞癌中存在有原发性耐药的现象，且多种机制并存。
This paper summarizes a variety of related molecules which are commonly used in HCC clinical diagnosis.
本文概述了临床常用的多种HCC诊断相关分子。
Table 5 depicts the molecular therapies currently tested within phase II and III clinical trials in HCC.
表5描述了目前检查为HCC的II期和II I期临床试验的分子治疗。
The FHIT protein deletion in HCC was higher than that in paratumor tissues and normal liver tissues (P<0.01).
正常肝组织中全部呈阳性表达，HCC组织中FHIT蛋白缺失率显著高于相应癌旁及正常肝组织（P<0.01）。
Conclusion: IL-17 is highly expressed in HCC tissues, which may provide a new target in the diagnosis and therapy of HCC.
结论：IL - 17在肝癌组织中高表达，有可能作为肝癌诊断及治疗的新靶点。
Objective To explore the possibility of tumor associated antigen encoded by MAGE-1gene used as a tarest for immunotherapy in HCC patients.
目的探讨MAGE-1基因编码的肿瘤相关抗原作为肝细胞性肝癌（HCC）主动免疫治疗攻击靶点的可能性。
Conclusion promoter methylation of CHFR and EDNRB genes is a common event in HCC, while frequency of promoter methylation of 3-ost-2 is rather low.
结论肝细胞癌中CHFR基因、EDNRB基因启动子的高甲基化是普遍现象，而3 -OST - 2基因启动子甲基化率较低。
The results indicate that changes in sugar chain structure of AFP in HCC patients do not result from increased or decreased activities of serum AFU.
提示，肝脏病变时afp糖链结构的变化不是血清afu参与的结果，可能来自于糖链生物合成途径的改变。
AIM: To study the specificity of the expression of PEG10 in HCC tissues and evaluate the feasibility for PEG10 as a novel molecular target of gene therapy for HCC.
目的：研究遗传印记基因PEG10在肝癌组织中表达的特异性，及在不同肿瘤细胞中的表达情况，为其作为一个潜在的肝癌特异标志物和HCC基因治疗的新的分子靶点提供实验依据。
Objective: to determine sialidase activity in human hepatocellular carcinoma (HCC) so as to probe into the mechanism of gangliosides change (GD3 increasing) in HCC.
目的：检测肝癌组织中唾液酸酶的活性，探讨肝癌组织中神经节苷脂谱改变(GD 3含量增加)的机理。
Hepatology Digest: there has been some progress in HCC screening with more patients being diagnosed early so there are many changes and challenges in clinical practice.
国际肝病：在HCC筛查方面已经取得了一些进展，更多的患者可以被早期诊断，因此在临床实践中有许多变化和挑战。
In addition, specific genes included in these signatures should be evaluated as potential targets for adjuvant treatment, following surgical intervention in HCC patients.
另外，对于经过手术治疗的患者中，基因标签内某些特定的基因也有望成为辅助治疗的靶点。
This review introduces the ultrasonic contrast agents, relative technologies, CEUS in HCC diagnosis, guidance of interventional treatment and assessment the curative effect.
本文介绍了超声造影剂及造影相关技术，超声造影在肝癌的诊断、指导介入治疗并评价疗效中的作用等方面内容。
The expression of the serum VEGF in HCC was significantly elevated as compared to the enhancement features and capsule(P<0.05), While no correlation with the size of HCC(P>0.05);
血清VEGF的表达与肝癌强化特征、包膜完整性均有较显著性差异（P<0.05），与肝癌的大小无显著性差异（P>0.05）；
The expressions of VEGF and EGF in HCC were not only correlated with the malignant behaviors including neovascularization and venous invasion, but also with the poor prognosis of HCC.
HCC组织中vegf禾egf的表达与HCC中新生血管生成、恶性生物学行为以及预后不良有密切关系。
He concludes that the study's findings support the basic principle behind reducing tumor size in HCC patients before transplant, because this may eliminate the most aggressive tumors.
该研究的结果支持在HCC患者进行肝脏移植前减小肿瘤大小的原则，因为这可能消灭最具有侵袭力的肿瘤；
CONCLUSION: CIK cell transfusion after micro-invasive treatments may improve the immunologic function in HCC patients, and play an important role in reducing the recurrence rate of HCC.
结论：微创治疗后联合cik细胞免疫治疗可提高肝癌患者的免疫功能，对降低肝癌的复发率具有重要作用。
Results There was no FAP-1 expression in normal controls and low positive expression rate in para-cancerous tissue (6.7%), but high positive expression rate (78.3%) was found in HCC (P<0.01).
FAP-1蛋白在正常肝组织中未见表达，癌旁肝硬化组织中的表达率为6.7%，而在恶性肿瘤中的阳性表达率分别为78.3%，与前两者比较差异有统计学意义（P<0.01）。
HCC shares fell as much as 6.3 percent in early trade but later recovered in the afternoon and were flat at 1330 GMT.
HCC的股票在早盘交易中降低了6.3%然后恢复，在下午格林尼治标准时间1：30持平。
Purpose To evaluate the combined imaging findings of spontaneous rupture and hemorrhage in hepatocellular carcinoma(HCC)and the predictive value.
目的探讨与肝癌自发破裂出血相关的影像学征象及其在预测中的价值。
To evaluate the diagnosis and differential diagnosis value of low-field MR plain scanning in hepatocellular carcinoma (HCC) and hemangioma.
目的探讨低场MR平扫在肝细胞癌(HCC)的诊断与鉴别诊断中的价值。
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