Results: the expression of hypoxic stress peptide was found and the level of cortisol increased in these patients.
结果：颅脑外伤后早期有低氧应激肽表达，皮质醇含量增高。
Objective: To investigate the expression of hypoxic stress peptide and cortisol in early period after craniocerebral trauma and its clinical significance.
目的：研究颅脑外伤后早期人体血清低氧应激肽效应表达及临床意义。
Conclusion Under hypoxic conditions, propofol can cause apoptosis in PC12 cells by inducing oxidative stress injury.
结论低氧环境下丙泊酚可通过氧化应激损伤导致PC 12细胞凋亡增加。
Conclusion: the endoplasmic reticulum stress-induced apoptosis may be one of the mechanism of hypoxic pulmonary hypertension and pulmonary vascular wall remodeling.
结论：内质网应激介导的凋亡与大鼠慢性低氧性肺动脉高压、肺血管改建的病理过程有关。
Conclusion: the endoplasmic reticulum stress-induced apoptosis may be one of the mechanism of hypoxic pulmonary hypertension and pulmonary vascular wall remodeling.
结论：内质网应激介导的凋亡与大鼠慢性低氧性肺动脉高压、肺血管改建的病理过程有关。
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