Ojective To establish a rat model of hyperoxia induced acute lung injury.
目的建立高氧性急性肺损伤大鼠模型并进行评价。
Objective: To demonstrate changes of KGF in premature rats with hyperoxia-induced CLD.
目的探讨高氧致CLD早产大鼠KGF表达的变化规律。
RA had a protective effect on hyperoxia lung injury by which declined AECII apoptosis and necrosis.
RA通过降低AECII凋亡、坏死从而对高氧肺损伤具有保护作用。
AIM: to investigate the anti-fatigue effect of hyperoxia solution injected to the caudal vein of mice.
目的：探讨小鼠尾静脉注射高氧液的抗疲劳效果。
AIM: To observe the therapeutic effects of hyperoxia solution orally or transvenously on hypoxemia in high altitude.
目的：探讨高氧液经静脉和消化道对高原低氧血症的作用。
Results After hyperoxia exposure, there were edema and inflammatory cell infiltration in the lung tissues of SD rats.
结果高氧暴露的SD大鼠肺组织可见水肿、大量炎症细胞浸润等急性炎症反应。
Objective: To discuss the protect effect of Tan Re-Qing injection on lung injury induced by hyperoxia in newborn rats.
目的：探讨痰热清注射液对新生大鼠高氧肺损伤的影响。
The proteasome inhibitor MG-132 can reduce the lung injury induced by hyperoxia and inhibit P38MAPK signaling pathway.
蛋白酶体抑制剂MG- 132可以减轻高氧引起的肺损伤，可能对p 38 MAPK信号通路有抑制作用。
Objective To study the effects of hyperoxia on ventilator-induced lung injury(VILI) and the protection of dexamethasone.
目的观察高浓度氧对通气机所致肺损伤的影响，以及地塞米松的防护作用。
Conclusion Early application of hyperoxia liquid can improve microcirculation, and decrease brain anoxia and brain edema.
结论早期使用高氧液可降低血液粘度，改善微循环，有助于改善脑组织缺血缺氧，减轻脑水肿。
Objective to study the pathologic alteration and oxidative stress in lung of neonatal rats exposed to hyperoxia for different time.
目的研究新生鼠暴露于高浓度氧不同时间后肺组织的病理改变，以及肺组织氧化应激反应状况。
Conclusion: the phosphorylation of RB protein can be induced by hyperoxia. Excessive proliferation of LF leads the pulmonary interstitial fibrosis.
结论：高氧可能导致RB蛋白磷酸化(RB灭活)，LF过度增殖最终导致肺间质纤维化。
Objective To investigate the protective effect of hyperoxia liquid on multiple organ damage during ischemic reperfusion injury in infants with muggy syndrome (MS).
目的探讨高氧液对捂热综合征(MS)患儿多脏器缺血-再灌注损伤的保护作用。
However, recent studies in animals have shown that prolonged exposure to an elevated level of oxygen, or hyperoxia, can cause long-term lung injury that resembles ARDS.
然而，最近的动物实验显示，长时间暴露在氧含量较高(氧过多)的环境可导致类似ARDS的肺长期损伤。
Objective: To explore the dynamic changes of the expression of VEGF protein in the lung and ultrastructure of pulmonary vascular endothelial cell in neonatal rats with hyperoxia.
目的探讨高浓度氧对新生鼠肺组织血管内皮生长因子(VEGF)蛋白表达及肺血管内皮细胞超微结构影响的动态变化规律。
Objective to investigate the expression of insulin-like growth factor-binding protein-5 (IGFBP-5) in full-term rats with hyperoxia-induced chronic lung diseases (CLD) and its mechanism.
目的研究胰岛素样生长因子结合蛋白5 (IGFBP - 5)在高体积分数氧致新生大鼠慢性肺疾病(CLD)中的表达及作用机制。
CONCLUSION Hyperoxia solution can mitigate the cerebral ischemia and reperfusion injury.
结论高氧液对神经元缺血损伤具有保护作用。
Objective: To discuss the protect effect of Urokinase injection and Tan re-qing injection on lung injury induced by Hyperoxia in newborn rats.
目的：探讨注射用尿激酶、痰热清注射液对新生大鼠高氧肺损伤的影响。
Objective: To discuss the protect effect of Urokinase injection and Tan re-qing injection on lung injury induced by Hyperoxia in newborn rats.
目的：探讨注射用尿激酶、痰热清注射液对新生大鼠高氧肺损伤的影响。
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