Mouse mutants play a key role in defining mammalian gene function and modeling human disease, and are of particular importance for analyzing and exploring disease pathogenesis.
小鼠突变对于展现哺乳动物基因功能和模拟人类疾病具有关键性的作用，对分析和探讨疾病的发病机制具有特别重要意义。
Receptor binding activity of the two mutants gives 66% and 54% of that of human proinsulin, respectively, while radio immuno activity drops to 22% and 9%.
两者的胰岛素受体活性分别为人胰岛素原的66%及54%，而胰岛素的放射免疫活性分别降为人胰岛素原的22%及9%。
Receptor binding activity of the two mutants gives 66% and 54% of that of human proinsulin, respectively, while radio immuno activity drops to 22% and 9%.
两者的胰岛素受体活性分别为人胰岛素原的66%及54%，而胰岛素的放射免疫活性分别降为人胰岛素原的22%及9%。
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