Objective To explore the role of oxygen free radicals (OFR) in hepatic preservation-reperfusion injury (HPRI) of rats.

目的探讨氧自由基在肝脏保存再灌注损伤(HPRI)中的作用。

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Results SOD activity decreased significantly (P<0.05), MDA and AST concentration increased remarkably (P<0.05, P<0.05), and abnormal morphological changes of hepatocytes were obvious during HPRI.

结果HPRI时，SOD活性显著下降（P＜0.05），MDA 含量和AST 活性明显升高（P＜0.05，P＜0.05），肝细胞形态发生异常变化。

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Results SOD activity decreased significantly (P<0.05), MDA and AST concentration increased remarkably (P<0.05, P<0.05), and abnormal morphological changes of hepatocytes were obvious during HPRI.

结果HPRI时，SOD活性显著下降（P＜0.05），MDA 含量和AST 活性明显升高（P＜0.05，P＜0.05），肝细胞形态发生异常变化。

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