The host immune response to Treponema pallidum is so complicated.
宿主对梅毒螺旋体的免疫反应比较复杂;
Moreover in the case of protease inhibitors there is also evidence that reconstitution of the host immune response will result in enhanced endogenous interferon production.
另外,在应用蛋白酶抑制剂时,有证据表明宿主免疫应答的重建可以加速内源性干扰素的合成。
However, recent progress to circumvent the host immune response in the brain has raised encouraging perspectives for intracerebral xenotransplantation as restorative strategy.
但是,最近在规避宿主脑部免疫排斥方面取得的进展,对于开展恢复性脑异种器官移植策略是一个极大的鼓舞。
As the main defense cell of the host immune response, neutrophils present lots of biological behaviors such as chemotaxis, phagocytosis, generating oxygen free radicals and so on.
作为宿主抵抗感染的主要防御细胞,中性粒细胞具有趋化、吞噬、产生氧自由基及释放作用等生物学特性。
After sacrifice, spinal cord sections were analyzed for MSC graft size, tissue sparing, host immune response, and glial scar formation, using specific antibodies and Nissl-myelin staining.
处死后,用特殊的抗体和髓鞘尼氏染色法处理脊髓切片,分析骨髓基质细胞移植物大小、组织保留情况、宿主免疫反应和神经胶质疤痕形成情况。
After sacrifice, spinal cord sections were histologically analyzed for presence of graft-derived immune cells, host immune response, tissue sparing, glial scar formation, and grafting efficacy.
处死后,进行脊髓切片病理分析移植性免疫细胞存在情况,宿主的免疫反应情况,组织保留情况,胶质瘢痕形成情况和移植治疗效果。
New research identifies a previously unknown enzymatic mechanism that subverts the early host immune response and promotes pathogenicity by manipulating a common signaling pathway in host cells.
新的研究发现了一种通过操纵宿主细胞内的一种常见信号通路来破坏早期宿主免疫反应并提高致病性的以前未知的酶反应机制。
Alveolar macrophages (AM), which are enhanced by GM-SCF, are an essential piece of the innate immune response and are known to contribute to host defense against flu infections in animal models.
肺泡巨噬细胞(AM)被GM - SCF强化,这是先天免疫应答的重要部分,已知在动物模型有助宿主防御流感病毒感染。
The host cells in question are immune-system cells called T-cells. Specifically, they are "helper" T-cells, which orchestrate the immune system's response to pathogens such as viruses.
被选中的寄主细胞是一种被叫做T细胞的免疫系统细胞,具体而言,他们是辅助T细胞[注一],它将应答免疫系统对例如病毒等病原体的反应。
After hyperthermia treatment of the tumor, the immune function of host 'body changes and the systemic anti-tumor immune response of host is activated.
肿瘤热疗后宿主机体的免疫功能发生变化,宿主全身抗肿瘤免疫反应被激活。
Preclinical studies have shown that PDT of Tumor auguments the host specific antitumor immune response.
许多动物实验表明,PDT治疗后,机体产生了特异性抗肿瘤免疫反应。
There was no apparent immune rejection and inflammatory response from the host tissue.
微囊化细胞周围无显著免疫排斥反应及炎症反应。
Macrophages play a significant role in the host defense mechanism. And it is the key participants in the innate immune response.
巨噬细胞在宿主防御反应中起了重要的作用,是天然免疫应答的主要参与者。
Macrophage is a central cell type in directing host inflammatory and immune processes; thus, its response to biomaterials is extremely important in understanding material-mediated host response.
巨噬细胞是调控宿主免疫和炎症反应的重要细胞,因此它对生物材料的响应性对认知材料-宿主反应有重要作用。
As a local therapeutic modality, photodynamic therapy (PDT) has been applied to various cancers. Preclinical studies have shown that PDT induces the host anti-tumor immune response.
光动力学疗法已用于多种肿瘤的局部治疗,研究表明光动力学疗法可诱导宿主产生抗肿瘤免疫应答。
Numerous studies have demonstrated that PDT is an effective therapy strategy. It enhances the host antitumor immune response, but the mechanisms behind this enhancement are unknown.
研究表明光动力学治疗是一种有效的肿瘤治疗方法,它可以引发特异性的抗肿瘤免疫作用,但具体的机制目前尚不清楚。
When a host is infected with the poxvirus, binding of A41L to cell surface proteins on the host cells results in suppression of the immune response.
当宿主感染痘病毒时,A41L与宿主细胞上的细胞表面蛋白的结合导致对免疫应答的抑制。
Lentinan enable host cells to generate antitumor immune response by stimulating and enhancing immunological function response, and can control and kill tumor cells.
香菇多糖通过非特异性地激发和增强机体的免疫功能,使机体产生抗肿瘤免疫应答,从而控制和杀灭肿瘤细胞。
Conclusion Both IL 2 and IL 12 are able to inhibit the growth of mice H22 hepatocellular carcinoma grafted subcutaneously and induce the host anti tumor immune response efficiently.
结论IL12、IL2基因治疗可抑制小鼠肝癌H 2 2皮下移植瘤的生长,提高机体的抗肿瘤免疫应答,两者联合运用可产生协同效应。
Conclusion Both IL 2 and IL 12 are able to inhibit the growth of mice H22 hepatocellular carcinoma grafted subcutaneously and induce the host anti tumor immune response efficiently.
结论IL12、IL2基因治疗可抑制小鼠肝癌H 2 2皮下移植瘤的生长,提高机体的抗肿瘤免疫应答,两者联合运用可产生协同效应。
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