Both simvastatin and CDPC can relieve the damages of the brain in rats HIBD.
辛伐他汀和胞二磷胆碱可减轻新生大鼠hibd后脑组织损害。
Objective To observe the electric physiologic changes in newborn piglets with HIBD.
目的观察缺氧缺血性脑损伤新生猪脑电生理的变化。
RESULTS: In the behavioral tests, HIBD rats performed significantly worse than control rats.
结果：在行为测试中，HIBD组大鼠的成绩显著低于正常鼠。
Objective To develop an improved neonatal piglet model of hypoxic ischemic brain damage (HIBD).
目的研究新生猪缺氧缺血脑损伤(HIBD)模型的制备。
Knock-down of endogenous protective process of MLT maybe plays a part in early HIBD pathogenic mechanism.
降低了MLT的内源性保护作用可能参与早期HIBD的发病机制。
Objective To evaluate treatment response of scopolamine for hypoxic-ischaemic brain damage(HIBD) in neonates.
目的探索东莨菪碱对新生儿缺氧缺血性脑损伤（HIBD）的治疗作用。
The hypoxic ischemic brain damage (HIBD) seen in survivors with prenatal or postnatal asphyxia is a major cause of disabilities.
新生儿缺氧缺血性脑损伤是人类致残的主要原因之一。
Objective To study the expression of ICAM 1 at gene transcription level in neonatal rats after hypoxic ischemic brain damage(HIBD).
目的研究新生动物缺氧缺血性脑损伤（HIBD）后脑细胞间粘附分子-1（ICAM- 1）在转录水平表达规律。
AIM To study the protective effects of monosialotetrahexosylganglioside (GM1) after hypoxic ischemic brain damage (HIBD) in neonatal rats.
目的研究外源性单唾液酸四己糖神经节苷脂（GM1）对新生鼠缺氧缺血脑损伤（HIBD）的保护作用。
Hypoxia ischemic brain damage (HIBD), which resulted from neonatal asphyxia, is the main cause of death and disability in neonatal periods.
围产期窒息所致缺氧缺血性脑损伤(HIBD)是新生儿死亡和残疾的主要原因。
Aim: To study the effects of epicranium medication injection treatment on learning and memory in brain cells of hypoxic-ischemic brain damage (HIBD).
目的：探讨头部药物位点注射对缺氧缺血性脑损伤(HIBD)后学习和记忆的影响。
Conclusion Both the CDPC and Shenmai injection can improve the learning and remembering ability of rat with HIBD, the later is better than the former.
结论：参麦注射液和胞二磷胆碱均可改善大鼠hibd后的学习和记忆能力，前者优于后者。
The GFAP increases in damaged cerebral cortex in the recovery stage of neonatal rat HIBD, suggesting that GFAP participates the repair of lesion region.
新生鼠hibd恢复期，脑损伤部位的GFAP明显增加，提示其可能参与了损伤区的修复。
Objective To observe the variations in the expression levels of interleukin-16(IL-16) protein in neonatal rats with hypoxic-ischemic brain damage(HIBD).
目的观察缺氧缺血性脑损伤（HIBD）新生大鼠白细胞介素-16（IL-16）表达的变化。
To explore the pathologic and DNA changes in the brain of the hypoxic ischemic brain damage (HIBD) newborn rats after exposed to high oxygen for long time.
观察缺氧缺血性脑损伤(HIBD)新生大鼠长时间吸高浓度氧后脑病理学及DNA损伤的变化。
In this review, we summarized relationship between apoptosis and neonatal hypoxic-ischemic brain damage (HIBD) and importance of apoptosis mechanism in HIBD.
该文主要阐述细胞凋亡与新生儿缺氧缺血性脑损伤的关系，提出细胞凋亡机制在缺氧缺血性脑损伤中的重要性。
Objective to study the repairing effects of nerve cells on HIBD with Epicranium Medication Injection treatment on site, and to explore the mechanism of action.
目的：研究头皮药物位点注射疗法对缺氧缺血性脑损伤(HIBD)后，神经细胞修复作用的疗效及探讨其作用机理。
Objective To study the repairing effects of epicranium medication injection treatment on brain cells of hypoxic-ischemic brain damage (HIBD) and its mechanism.
目的研究头皮药物位点注射对缺氧缺血性脑损伤(HIBD)后脑细胞的修复作用。
Objective to explore the influences of early interventions on learning, memory and neuron apoptosis in the brain of neonatal rats with hypoxic ischemic brain damage (HIBD).
目的探讨早期干预对缺氧缺血性脑损伤(HIBD)大鼠学习记忆能力及神经元凋亡的影响。
Conclusion The role of environmental stimulation in functional outcome of HIBD is likely to be related to change of synaptic substructure and increased synaptic plasticity.
结论环境刺激对HIBD大鼠功能的促进作用可能与突触超微结构参数的改变，增强突触可塑性有关。
Objective: To study the effect of the neonatal handling and enriched environmental stimulation on brain function of rats with hypoxic-ischemic brain damage (HIBD) in uterus.
目的：探讨早期触摸和环境刺激对宫内缺血缺氧性脑损伤(HIBD)大鼠脑功能的影响。
Objective To study the role of endogenous carbon monoxide(CO) in newborn rats with hypoxic-ischemic brain damage (HIBD) and the protective effect of Sn-protoporphyrin (SnPP).
目的探讨内源性一氧化碳（CO）在新生大鼠缺氧缺血性脑损伤（HIBD）中的作用机制及锡原卟啉的保护作用。
Objective: To study the changes of pathology, ATP production rate and ATP synthase activity in hippocampal cortex of neonatal pigs after hypoxic-ischemic brain damage (HIBD).
目的：研究缺氧缺血性脑损害新生猪海马皮层病理改变及ATP生成率、ATP合成酶活性变化。
Objective To investigate the changes of blood coagulation state and some prothrombin's function in newborn piglets at different time after hypoxic-ischemic brain damage (HIBD).
目的研究缺氧缺血新生猪凝血状态，以及部分凝血因子促凝活性的动态变化，探讨缺氧缺血性脑损伤（HIBD）时凝血功能变化。
Objective To study the neuroprotective effects of lamotrigine (LTG) on hypoxic-ischemic brain damage (HIBD) in neonatal rats, and the influence of administration time and dosage.
目的研究拉莫三嗪(ltg)对缺氧缺血性脑损伤(HIBD)新生大鼠的神经保护作用，以及不同用药剂量和用药时间对该作用的影响。
Conclusions Pretreatment with baicalin, as with Dex, has a protective effect against HIBD in neonatal rats, but baicalin or Dex post-treatment do not reverse the neuronal injuries.
结论黄芩苷及地塞米松预处理对新生大鼠hibd产生保护作用，能减少神经元凋亡，而后处理无效。
Objective To observe the change of neuron-specific enolase (NSE) and glial fibrillary acidic protein (GFAP) in neonatal rats' cerebral cortex with hypoxic-ischemic brain damage (HIBD).
目的了解新生鼠缺氧缺血性脑损伤(HIBD)时大脑皮质神经元特异性烯醇化酶(nse)和胶质纤维酸性蛋白(GFAP)的变化。
Through investigating the effect of mild hypothermia on activity of nitric oxide synthase(NOS) in cortical neurons and glycemia levels of neonatal rats with hypoxic ischemic brain damage(HIBD).
通过亚低温对新生鼠缺氧缺血性脑损伤（HIBD）大脑皮质神经元一氧化氮合酶（NOS）及血糖水平影响的研究，探讨亚低温对缺氧缺血性脑损伤的保护作用机制。
Objective to observe the expression of proliferation in hippocampus following hypoxic-ischemic brain damage (HIBD) in neonatal rats and to explore the possible endogenous neurogenesis of brain.
目的：观察新生大鼠缺氧缺血性脑损伤(HIBD)后海马区细胞增殖的情况，探讨脑组织内源性修复的可能机制。
Objective to study the morphologic changes of mitochondria and metabolic protective effect of fructose 1 6 diphosphate (FDP) intervention in newborn rats with hypoxic ischemic brain damage (HIBD).
目的探讨16二磷酸果糖(FDP)干预治疗新生鼠缺氧缺血性脑损伤(HIBD)后脑神经细胞线粒体形态学变化及对能量代谢的影响。
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