Objective To study the protection of ischemic preconditioning on the hepatic ischemia-reperfusion injury.
任智目的评价缺血预处理对大鼠肝脏的保护作用。
Conclusions The apigenin have a prominent protective effect on hepatic ischemia reperfusion injury in rats.
结论芹菜素对大鼠肝脏缺血再灌注损伤有一定的保护作用。
Apoptosis is an important pathophysiological characteristic during hepatic ischemia-reperfusion injury (HIRI).
细胞凋亡是肝缺血—再灌注损伤重要的病理生理特征。
Objective To study the protection of ischemic preconditioning (IPC) on the hepatic ischemia reperfusion injury.
目的评价缺血预处理（IPC）对大鼠肝脏的保护作用。
It was established the canine model of whole hepatic ischemia-reperfusion with blocked hepatoduodenal ligament.
阻断肝十二指肠韧带建立犬全肝缺血再灌注模型。
Objective To explore the protective effect of atomolan in treating hepatic ischemia and reperfusion(I/R) injury.
目的探讨阿拓莫兰对家兔肝缺血再灌注（I/R）损伤肝脏的保护作用。
Objective: To explore the protective effects of Puerarin injection on hepatic ischemia-reperfusion injury (HIRI).
目的：观察葛根素注射液对家兔肝缺血再灌注损伤(HIRI)的防治作用。
Objective To investigate the protective effect and its role of apigenin on hepatic ischemia reperfusion injury in rats.
目的观察芹菜素对大鼠缺血再灌注肝脏损伤的防护作用。
Objective To investigate the protective effect of ischemia postconditioning on hepatic ischemia-reperfusion injury in rabbits.
目的探讨缺血后处理对肝脏热缺血再灌注损伤的保护作用。
Objective:To investigate the effectiveness of dietary nitrite supplements on hepatic ischemia-reperfusion(I/R) injury in rats.
目的：探讨饮食中亚硝酸盐对大鼠肝脏缺血再灌注（I/R）损伤的保护作用。
Objective To investigate the protective effect of zinc on hepatic ischemia reperfusion injury and discuss the possible mechanism of it.
目的研究锌对肝缺血再灌注损伤的保护作用，并探讨其可能的机制。
Objective: To evaluate the role of oxygen free radicals in hepatic ischemia and reperfusion injury (HIRI) and the influence of tiopronin.
目的：探讨氧自由基在肝缺血再灌注损伤(HIRI)中的作用及凯西莱的防护作用。
Objective: To investigate the effects of Aspirin DL Lysine on hepatic ischemia reperfusion injury in the rats and its mechanism of action.
目的：观察黄芪注射液对大鼠肾脏缺血再灌注细胞凋亡的影响，探讨其对肾脏缺血再灌注损伤的保护机制。
Objective: To study the effect of Psoralea Codrylifolia L. extract on Superoxide dismutase(SOD) activities in mice damaged by hepatic ischemia reperfusion.
目的：探讨补骨脂注射剂对肝缺血再灌注损伤超氧化物歧化酶活力（SOD）的影响。
Objective: To study the role of lipid peroxide reaction in hepatic ischemia damage and the protective effects of antioxidants, ligustrazine and coenzyme Q10 (co -q10).
目的：探讨脂质过氧化反应在门腔分流术后肝脏缺血损害中的作用及抗氧化剂川芎嗪，辅酶Q 10的保护作用。
Objective: to observe the effects of hepatic ischemia reperfusion on cell cycle, and to investigate the probable causes of the poor liver function after transplantation.
目的：观察肝缺血再灌注对细胞周期的影响，以探讨移植肝功能不良的可能原因。
Conclusions: Epidural anesthesia combined with isoflurane can reduce the stress response and produce protective effects on the hepatic ischemia-reperfusion injury to some extent.
结论：硬膜外阻滞复合异氟烷吸入，可减轻手术应激反应，对肝脏缺血再灌注损伤有一定的保护作用。
Conclusion Inhibiting activation of Kupffer cells may attenuate hepatic ischemia reperfusion injury, and Kupffer cells may play an important role in hepatic ischemia reperfusion injury.
结论抑制枯否细胞活化可减轻肝脏缺血再灌注损伤，枯否细胞在肝脏缺血再灌注损伤中的作用很重要。
Research background and Object: Hepatic ischemia-reperfusion injury is one of the main cause which induces the graft dysfunction during operation or post-operation of liver transplantation.
研究背景及目的：肝移植术中术后移植肝缺血-再灌注损伤是导致肝移植失败的主要原因之一。
Conclusion Nitric oxide could increase the release of the hepatic enzymes after liver ischemia reperfusion injury, inhibit the apoptosis of hepatocytes and improve liver ischemia reperfusion injury.
结论一氧化氮可减少肝缺血再灌注损伤后肝酶的释放，抑制肝细胞的凋亡，改善肝缺血再灌注损伤。
Conclusion In the process of liver transplantation, the ischemia reperfusion damage may lead to hepatic microcirculation disturbance, which is a major cause of graft failure.
结论肝移植过程中，缺血再灌注损伤可造成肝脏微循环紊乱，是供肝失活的一个重要原因。
The method of liver resection in normothermic first hepatic intermittent ischemia is technically simple, less bleeding and easy to extent.
常温下第一肝门间歇阻断切肝法操作简单，出血少，易于推广。
To discuss the law of the liver metabolism before and after the hepatic inflow occlusion and the mechanism of the ischemia-reperfusion injury.
探讨肝血流阻断前后肝脏的部分代谢规律及缺血再灌注损伤的机制。
Conclusion Hepatic energy metabolic function is reversible in rats with portal vein ischemia within 90 minutes but irreversible in those with 120 minutes of portal vein ischemia.
结论在门静脉转流下大鼠门静脉缺血90 min以内肝脏能量代谢损害可逆，而门静脉缺血120min则不可逆。
Using a rat model of hepatic warm ischemia-reperfusion, the hepatic histomorphology, enzymology, lipid peroxidation, apoptosis and the plasma level of Endothelin-1 were observed.
采用大鼠肝脏热缺血再灌注模型，观察肝脏组织形态学、酶学、脂质过氧化、细胞凋亡及血浆内皮素的变化。
Studies have shown that silibinin also has a good treatment of hyperlipemia, eliminating free radicals, anti-hepatic lipid peroxidation, anti-ischemia-reperfusion injury.
研究表明，水飞蓟宾还具有良好的治疗高血脂症、消除自由基、抗肝脂质过氧化作用、抗缺血再灌注损伤作用。
Studies have shown that silibinin also has a good treatment of hyperlipemia, eliminating free radicals, anti-hepatic lipid peroxidation, anti-ischemia-reperfusion injury.
研究表明，水飞蓟宾还具有良好的治疗高血脂症、消除自由基、抗肝脂质过氧化作用、抗缺血再灌注损伤作用。
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