• Conclusion: There is decline of HLA-ABC antigen expression in HCC tissues and HCC cell lines.

    结论肝癌组织肝癌细胞系中HLA - ABC抗原表达有所下降。

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  • Conclusion: IL-17 is highly expressed in HCC tissues, which may provide a new target in the diagnosis and therapy of HCC.

    结论IL - 17肝癌组织中高表达有可能作为肝癌诊断治疗靶点

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  • AIM: To study the specificity of the expression of PEG10 in HCC tissues and evaluate the feasibility for PEG10 as a novel molecular target of gene therapy for HCC.

    目的研究遗传印记基因PEG10肝癌组织表达特异性在不同肿瘤细胞中的表达情况,作为一个潜在的肝癌特异标志物和HCC基因治疗新的分子靶点提供实验依据。

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  • Immunohistochemistry was used to determine the expression of VEGF, MMP-2 in 60 HCC tissues and cancer adjacent tissues, and microvessel density (MVD) was detected with labelled CD34.

    采用免疫组化检测60例肝癌组织肝组织中vegf及MMP - 2表达CD 34标记免疫组化法检测微血管密度(MVD)。

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  • CONCLUSIONS: We developed a composite prognostic model for HCC recurrence, based on gene expression patterns in tumor and adjacent tissues.

    结论根据肿瘤及其邻近组织基因表达特征我们建立了一个HCC复发复合预后模型

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  • Objective to study the relationship between the intercellular adhesion molecule-1 (ICAM 1) expression of hepatocellular carcinoma (HCC) tissues and its invasion and metastasis.

    目的研究肝癌组织中细胞粘附分子-1 (ICAM - 1)的表达肝癌侵袭转移关系

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  • To establish a set of two-dimensional polyacrylamide gel electrophoresis (2-DE) techniques for proteomic research of hepatocellular carcinoma (HCC) tissues.

    优化双向凝胶电泳实验步骤,总结出适用原发性肝癌组织的双向凝胶电泳技术

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  • Aim: to study the expression of P16 in tissues of primary hepatocellular carcinoma (HCC).

    目的探讨原发性肝癌(HCC)P 16蛋白表达

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  • The FHIT protein deletion in HCC was higher than that in paratumor tissues and normal liver tissues (P<0.01).

    正常组织全部呈阳性表达,HCC组织中FHIT蛋白缺失率显著高于相应旁及正常肝组织(P<0.01)。

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  • STAT3, which is associated with inflammation and cellular transformation, is activated in human HCC tissue but not in normal human liver tissues.

    STAT3联系炎症细胞转化的桥梁,肝癌组织活化正常肝脏没有类似表现。

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  • STAT3, which is associated with inflammation and cellular transformation, is activated in human HCC tissue but not in normal human liver tissues.

    STAT3联系炎症细胞转化的桥梁,肝癌组织活化正常肝脏没有类似表现。

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