Under drought stress, NO and H2O2 probably exerted their regulating effect on ABA synthesis by regulating cellular redox status.
干旱胁迫下NO和H2O2对ABA合成的调节作用可能是通过调节细胞氧化还原状态进行。
In order to explore the relationship between stress of harmful factors and SO2 biological synthesis in cells, biosynthesis of SO2 induced by H2O2 at various concentrations 0.
为了探讨有害因子胁迫与二氧化硫SO2细胞生物合成的关系，采用体外培养实验方法研究了不同浓度h2o2 0。
NO and H2O2 could lead to the cellular transition towards oxidative status by simulating the effect of drought stress, and they could also increase ABA accumulation in wheat leaves.
NO和H2O2能模拟干旱胁迫的作用使细胞状态向氧化型转变，还可以使小麦叶片aba积累量上升。
Objective To investigate the protective effect of gross saponins of tribulus terrestris(GSTT) on H2O2-induced oxidative-stress damage in PC12 cells.
目的探讨蒺藜皂苷（GSTT）对H2O2诱导的PC12细胞氧化应激损伤的保护作用。
Several HO. scavengers significantly inhibited hemo lysis induced by hemin and ascorbic acid. It was concluded that H2O2 was involved in the oxidative stress mediated by hemin and ascorbic acd.
氢氧自由基淬灭剂显著地抑制抗坏血酸增强氯高铁血红素诱导红细胞溶血。由上述可得到如下结论：抗坏血酸增加氯高铁血红素诱导的红细胞氧化压力与H2O2有关；
Conclusion SBP could protect endothelial cells and it might be related to inhibit oxidative stress induced by H2O2.
结论麝香保心丸保护血管内皮细胞可能与抑制H2O2诱导人脐静脉内皮细胞氧化应激有关。
Conclusion SBP could protect endothelial cells and it might be related to inhibit oxidative stress induced by H2O2.
结论麝香保心丸保护血管内皮细胞可能与抑制H2O2诱导人脐静脉内皮细胞氧化应激有关。
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