"If this test could predict the onset of GVHD before the rash develops, it could be extremely valuable, " he says.
他说：“如果这种检测能够在皮疹显现之前预测移植物抗宿主病的发病，那将是非常有价值的。”
Acute GVHD was not observed in both groups.
二组病人皆未观察到急性GVHD。
TA-GVHD is rare, but its mortality is more than 90%.
本病虽少见但其病死率高达90 %以上。
Results Compared to B6 WT group, B6 CCR5 KO group succumbed to acute GVHD at an accelerated rate.
结果与B6WT组比较，B6CCR5KO组小鼠以更快的速度死于急性移植物抗宿主疾病（GVHD）；
"If this test could predict the onset of GVHD before the rash develops, it could be extremely valuable," he says.
他说：“如果这种检测能够在皮疹显现之前预测GVHD的发病，那将是非常有价值的。”
GvHD may be mild or life threatening, depending on the severity of reaction and the part of the body affected.
GVHD症状可以是轻微的，但也可以危及生命，这取决于排斥反应的严重性以及发生排斥反应的机体部位。
The liver, skin and intestine are the classic GVHD target organs and lungs may be a potential GVHD target organ.
肝、肠、皮肤是公认的移植物抗宿主病（GVHD）靶器官，肺可能也是潜在的GVHD靶器官。
The more T lymphocytes transfused, the higher the incidence of TA-GVHD, and the higher the mortality it induces.
输入的供者淋巴细胞数量越多，TA-GVHD的发生率越高，病情越重，死亡率也越高。
Elafin is produced in the surface layer of skin cells in response to specific inflammatory proteins involved in GVHD.
弹力素是由GVHD引发特异性炎症的皮肤表层皮肤细胞分泌。
After studying 500 patients with rashes, researchers found high levels of elafin only in those with rashes from GVHD.
通过研究500位发生皮疹的患者，研究者发现只有那些由于GVHD引发的皮疹会导致升高的弹力素。
Objective To develop transfusion associated graft vs host disease (TA-GVHD) model by infusing allogeneic splenocytes.
目的探讨输注同种脾细胞建立小鼠输血相关的移植物抗宿主(TA -GVHD)模型及其特点。
GvHD occurs when the immune cells from the donated blood-forming cells (graft) attack the body of the patient (host).
当来自供体造血细胞(移植物)的免疫细胞抨击袭击病人(宿主)的机体时便发生GVHD。
GVHD was assessed by signs of weight loss, ruffled fur, diarrhea and histologic changes of skin, liver and small intestines.
通过体重改变、弓背、翘毛、腹泻等表现和肝脏、小肠、皮肤病理学检查观察GVHD ；
It is suggested that Murine GVHD model successfully established can be used in evaluating the effects of anti GVHD therapies.
此结果表明，已成功地建立了小鼠gvhd模型，可用于评价抗gvhd的治疗效果。
Apart from relapse of a person's disease, GVHD is the principal cause of death in patients who have received donated bone marrow.
除了患者病情复发，移植物抗宿主病是接受骨髓细胞移植病人死亡的主要原因。
We have constituted a mouse model for fetal blood transplantation histocompatibility complex (MHC) without causing serious GVHD.
本室已经建立了MHC不相合单份鼠胎血同种移植的小鼠模型。
The test could predict GVHD before clinical symptoms develop, the researchers emphasize, which could allow for early intervention.
研究者强调，该检查能在出现临床症状前预测GVHD，从而早期干预。
Conlusion The mouse model of chronic GVHD, which is alike to human lupus nephritis, can be used as a good model of lupus nephritis.
结论慢性移植物抗宿主模型类似人类狼疮性肾炎，是良好的狼疮肾炎模型。
Objective To review the current condition, test method and progress of the animal model of xenograft versus host disease (xeno GVHD).
目的综述异种移植物抗宿主反应(GVHD)动物模型的研究现状、检测方法及进展情况。
Many researchers have therefore sought to develop techniques to prevent GVHD that avoid the complications incurred by immunosuppression.
因此许多学者已经在寻找开发一些防止GVHD同时避免免疫抑制引起的并发症的方法。
Objective To explore the role of TJU103 in preventing graft-versus-host disease (GVHD) after allogeneic stem cell transplantation in mice.
目的探讨TJU103对小鼠同种异基因造血干细胞移植模型移植物抗宿主病(GVHD)是否起预防作用。
Most (90%) clinical symptoms of GVHD develop 14 or more days after transplant, while this blood test is taken 7 days after the transplant.
GVHD的大部分临床症状(90%)始于移植后的14天或更久，但移植后7天已进行血检。
Is the GVHD more severe in the allogeneic peripheral blood stem cell transplantation compared with allogeneic bone marrow transplantation?
异基因外周血干细胞移植中的GVHD比骨髓移植更严重吗？
Conclusion Sensitized donor lymphocytes infusion after HSCT evidently promoted the immune reconstitution and reduced the incidence of GVHD.
结论造血干细胞移植后输注致敏供者的淋巴细胞能促进受者的免疫功能重建，并可减少GVHD的发生。
After 12 months, three times as many patients in the high-elafin group had died from GVHD and its complications than in the low-elafin group.
12个月之后，第一组移植物抗宿主病死亡率和并发症发病率是第二组的三倍。
After 12 months, three times as many patients in the high-elafin group had died from GVHD and its complications than in the low-elafin group.
12个月之后，第一组移植物抗宿主病死亡率和并发症发病率是第二组的三倍。
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