Chronic exposure to Cd mainly causes the renal proximal tubule damage, which represents that the excretion of proteinuria, glucosuria and aminoaciduria.
慢性接触镉主要引起肾小管损伤，表现为蛋白尿、糖尿和氨基酸尿。
Genetic defects of SGLT2 result in a benign familial renal glucosuria.
SGLT2的遗传缺陷会导致良性家族性肾性葡萄糖尿症。
However, stress - induced hyperglycemia does not usually in glucosuria (elevated urine glucose).
然而，应激诱导性高血糖并不常会造成尿糖升高。
Objective: To investigate the mechanism of stress glucosuria and renal function impairment in early stage of severe bum.
目的：通过对严重烧伤病人进行应激性糖尿症和早期肾功能损害等并发症观察研究，进一步了解其发病机制。
Conclusion: Glucosuria in part of the patients may arise from re - absorption dysfunction of renal proximal convoluted tubules.
结论：部分严重烧伤病人早期糖尿是由肾脏近曲小管吸收障碍引起。
Chronic exposure to Cd mainly causes the renal proximal tubule damage, which represents that the excretion of proteinuria, glucosuria, aminoaciduria, enzymaticuria and urinary cadmium increase.
慢性接触镉主要引起肾小管损伤，表现为蛋白尿、糖尿、氨基酸尿、酶尿及尿镉排出量明显增加等。
Chronic exposure to Cd mainly causes the renal proximal tubule damage, which represents that the excretion of proteinuria, glucosuria, aminoaciduria, enzymaticuria and urinary cadmium increase.
慢性接触镉主要引起肾小管损伤，表现为蛋白尿、糖尿、氨基酸尿、酶尿及尿镉排出量明显增加等。
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