The results indicated that there were glucocorticoid receptors on SPM.
结果提示脑突触质膜上存在糖皮质激素膜受体。
The effect was thought to be mediated via trabecular meshwork glucocorticoid receptors.
这种效应被认为是通过小梁网的糖皮质激素受体介导的。
Due to the presence of glucocorticoid receptors in almost all cells, both the desired and undesired effects of glucocorticoid therapy are manifold.
由于糖皮质激素受体的存在，几乎所有的细胞，无论是理想和不受欢迎的糖皮质激素治疗的影响是多方面的。
Conclusions Glucocorticoid receptors are down-regulated in CD3 + t lymphocytes in association with increased adrenal secretion of cortisol in ACLF.
结论慢加急性肝衰竭患者外周血cd3 +T淋巴细胞糖皮质激素受体的下调，与患者升高的皮质醇有关。
Objective to investigate the relationship between the contents of glucocorticoid receptors (GCR) in myasthenia gravis and the curative effects of steroid therapy.
目的探讨重症肌无力患者外周血白细胞糖皮质激素受体含量与应用糖皮质激素治疗效果的关系。
Objective to explore the relationship between the contents of glucocorticoid receptors (GR) in multiple sclerosis (MS) patients and the efficacy of steroid therapy.
目的探讨多发性硬化(MS)患者外周血白细胞糖皮质激素受体(GR)含量与糖皮质激素的治疗效果关系。
It has been hypothesized that changes in the glucocorticoid receptors in certain regions of the brain (eg, the hippocampus) might contribute to the depressive symptoms.
到目前为上的假说是：脑内某些区域如海马中糖皮质激素受体的改变可能引起抑郁症状。
Dexamethasone, a synthetic glucocorticoid, is believed to bind to the glucocorticoid receptors located in the anterior pituitary, thereby reducing ACTH secretion, and ultimately cortisol production.
地塞米松这种人工合成的糖皮质激素，被认为在与位于垂体前叶的糖皮质激素受体结合后使ACTH分泌减少，从而最终导致皮质醇产生减少。
Dexamethasone, a synthetic glucocorticoid, is believed to bind to the glucocorticoid receptors located in the anterior pituitary, thereby reducing ACTH secretion, and ultimately cortisol production.
地塞米松这种人工合成的糖皮质激素，被认为在与位于垂体前叶的糖皮质激素受体结合后使ACTH分泌减少，从而最终导致皮质醇产生减少。
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