Here we reviewed the pathomorphology and pathologic mechanism of glaucomatous damages in the central visual system.
我们对近年来青光眼中枢损伤的病理形态学及病理机制研究作一综述。
Patients with sickle cell disease or trait are particularly susceptible to glaucomatous optic nerve damage from even mildly elevated IOPs.
对于患有镰状细胞贫血的患者而言，即使是轻微的IOP升高，也很有可能造成青光眼性视神经损伤。
Objective Tostudy how the cup changed in depth and to assess the value the cup depth related parameters in diagnosis of glaucomatous neuropathy.
目的探讨青光眼视杯深度变化规律及视杯深度在青光眼诊断中的作用。
Glucocorticoid effects on the human trabecular meshwork can be used as a model system in which to study glaucomatous damage to the trabecular meshwork.
糖皮质激素对小梁网的作用可作为一个良好模型用于研究青光眼的发病机制。
ObjectiveTo discuss the clinical effect of the combination of lamellar keratoplasty and cyclocryotherapy for the treatment of glaucomatous bullous keratopathy.
目的探讨板层角膜移植联合睫状体冷凝术治疗青光眼性大泡性角膜病变的临床效果。
Three essential elements of glaucomatous optic nerve damages are optic nerve rim loss, retinal nerve fiber layer defects (RNFLD) and optic disc linear hemorrhages.
诊断青光眼视神经损害的三要素为盘沿丢失、视网膜神经纤维层缺损(RNFLD)及视盘线状出血。
Conclusion The enhanced level of anti-HSP27 antibody in serum with IOP increase in experimental rat glaucoma model might play an important role in glaucomatous optic neuropathy.
结论伴随眼压升高出现的血清中hsp27抗体水平升高可能在青光眼视神经损害中具有重要作用。
The article explains the glaucomatous definition from the five positions:intraocular pressure, fundus, visual field, the damage of glaucomatous opticus and the safeguard of opticus.
本文从眼压、眼底、视野、青光眼视神经损害机制以及视神经保护五个方面对青光眼定义进行了阐述。
The two trials that enrolled participants with pigment dispersion syndrome at baseline reported the proportion of participants with onset of glaucomatous visual field changes during the study period.
两个纳入的色素播散综合症受试者试验，报告在研究进行间发现受试者发生青光眼视野改变的比例。
The two trials that enrolled participants with pigment dispersion syndrome at baseline reported the proportion of participants with onset of glaucomatous visual field changes during the study period.
两个纳入的色素播散综合症受试者试验，报告在研究进行间发现受试者发生青光眼视野改变的比例。
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