Conclusion TET's poisonous effects do exert by means of antagonize the GABAA receptor.
结论TET是通过拮抗gabaa受体而发挥毒理效应的。
This model implicates GABAA-receptor dysfunction in patients as a causal predisposition to anxiety disorders.
这个模型暗示了在病患身上GABAA受体的官能障碍，作为一种焦虑症的病因体质。
GABAA receptor is the major neurotransmitter system in the central nervous system (CNS) and elicits a wide range of neuronal physiological activities.
GABAA受体是中枢神经系统内重要的抑制性受体，有广泛的神经生理活性。
Results: GABA uptake inhibitor nipecotic acid-induced increase of MAP was blocked by GABAA receptor antagonist bicuculline, but not by GABAB antagonist CGP 35348;
结果：GABAA受体拮抗剂荷包牡丹碱能逆转GABA摄取抑制剂3-哌啶甲酸引起的血压升高，GABAB受体拮抗剂CGP 35348则不能；
The previous studies indicated it produced anesthetic effect through its mimic GABA action, which enhanced central inhibition mediated by postsynaptic GABAa receptors.
以往研究认为其具有拟gaba样作用，通过增强突触后gaba_a受体介导的中枢抑制而产生麻醉作用。
Aim: to explore the influence of GABAergic neurotransmitters and GABAA receptors on the auditory afferent impulses recorded in the brainstem evoked by electro-stimulation.
目的：旨在探讨脑干听觉传入通路中gaba能神经递质及GABAA受体对电刺激位听神经传入冲动的影响。
Conclusion: Melatonin does not exhibit its potentiation sleeping time in mice through melatonin 3 receptor. Hypnotic activity of melatonin may be mediated through picrotoxin site on GABAA receptor.
结论：褪黑素延长小鼠睡眠时间的作用与褪黑素3型受体无关，而与GABAA受体关系密切，其作用主要由印防己毒素结合位点介导。
Conclusion: Melatonin does not exhibit its potentiation sleeping time in mice through melatonin 3 receptor. Hypnotic activity of melatonin may be mediated through picrotoxin site on GABAA receptor.
结论：褪黑素延长小鼠睡眠时间的作用与褪黑素3型受体无关，而与GABAA受体关系密切，其作用主要由印防己毒素结合位点介导。
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