However, the functional plasticity, accompanying the molecular modifications, remains to be elucidated for the understanding of the hippocampus-dependent information storage and memory retrieval.
然而,因分子特性改变造成的功能变化对于我们正确理解海马依赖性的信息储备和记忆提取的机制仍然非常局限。
Objective To estimate environmental influence on immature brain plasticity, and its role in functional outcome of brain damage in rats.
目的了解环境刺激对未成熟脑可塑性的影响及其在脑损伤后功能康复中的意义。
It is indicated that NCAM and GDNF may be involved in the changing process of cerebral plasticity after cerebral ischemia injury, also associated with the functional recovery.
提示神经细胞黏附分子和胶质细胞源性神经营养因子可能参与了脑缺血损伤后脑的可塑性变化过程且与功能恢复有关。
Conclusion The role of environmental stimulation in functional outcome of HIBD is likely to be related to change of synaptic substructure and increased synaptic plasticity.
结论环境刺激对HIBD大鼠功能的促进作用可能与突触超微结构参数的改变,增强突触可塑性有关。
This suggested that NMDA receptor must play a role in the plasticity of brain auditory functional development.
推测NMDA受体与脑听觉功能发育及其可塑性相关。
The neurons form connections and self-assemble into functional networks, allowing us access to the same phenomena of plasticity that occur in vivo.
这些神经元在培养皿中形成连结并自我聚集成功能性网络,让我们能观察到与活体中相同的可塑性形成现象。
Yet, while we are beginning to understand the cellular mechanisms underlying synaptic changes, it is important to consider the functional implications of synaptic plasticity in the intact brain.
当开始了解这些突触改变的细胞生物学机制的时候,也应该考虑这种突触可塑性在完整大脑中的功能意义。
The functional relation between the plasticity parameters and the strain rate was obtained according to the experimental data in the reference.
根据文献的实验数据,导出了塑性参数与应变率之间的函数关系。
The functional relation between the plasticity parameters and the strain rate was obtained according to the experimental data in the reference.
根据文献的实验数据,导出了塑性参数与应变率之间的函数关系。
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